Quantitative evaluation of testicular biopsies from men with unilateral torsion of spermatic cord

Chakraborty, J.; Hikim, Amiya P. Sinha; Jhunjhunwala, Jagadish S.

doi:10.1016/0090-4295(85)90531-x

Cited by 32 publications

(13 citation statements)

References 18 publications

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“…Pathologies deteriorating the ipsilateral testis such as torsion [1, 2]nondescent [3, 4], varicocele [5], incarcerated inguinal hernia [6, 7, 8]and vas deferens obstruction [9]may also cause adverse effects on the contralateral testis.…”

Section: Introductionmentioning

confidence: 99%

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DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

Salman¹,

Okur

Tanyel³

1998

European Urology

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Section: Introductionmentioning

confidence: 99%

“…Preexisting congenital defects [1, 2], incidental occurrence of other conditions such as varicocele [9]and autoimmunity [10]have been the most commonly incriminated mechanisms to explain the contralateral testicular damage.…”

Section: Introductionmentioning

confidence: 99%

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

Salman¹,

Okur

Tanyel³

1998

European Urology

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“…Incidental occurrence of other conditions, such as variocele and recurrent subclinical attacks of torsion in the contralateral testis, have also been proposed [13]. However, the presence of similar conditions cannot fully explain the contralateral testicular injury in various animals following experimental ipsilateral testicular torsion.…”

Section: Discussionmentioning

confidence: 99%

“…A preexisting congenital defect, subclinical attacks of contralateral testicular torsion, concurrence of contralateral pathology such as variocele and autoimmunity have all been proposed to account for the contralateral testicular damage [12, 13, 14]. However, none of the proposals is thought to fully explain the mechanism of contralateral testicular injury.…”

Section: Introductionmentioning

confidence: 99%

Hemodynamic Monitoring of the Contralateral Testis during Unilateral Testicular Torsion Describes the Mechanism of Damage

Salman

Mutlu

et al. 1998

European Urology

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“…Introduction defects in spermatogenesis have been proposed to account for contralateral testicular injury [8][9][10], but none has Because fertility problems are clinically demonstrable among men who previously experienced unilateral testicubeen universally accepted [11][12][13]. A current theory of contralateral testicular injury lar torsion (UTT), contralateral testicular injury after UTT has been studied extensively.…”

mentioning

confidence: 99%

Histological evidence of decreased contralateral testicular blood flow during ipsilateral testicular torsion

Ciftci¹,

Müftüoğlu

Çakar

et al. 1997

British Journal of Urology

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Objective To evaluate contralateral testicular blood flow eral testes of the torsion group were collapsed. The diÂerence between the contralateral testes of the two by histological examination of arterioles during ipsilateral testicular torsion.groups and between the ipsilateral and contralateral testes in the torsion group were significant. The diamMaterials and methods The study comprised two groups of 20 male albino rats (weight 250-270 g). The eter of uncollapsed arterioles did not diÂer significantly among either testes of the control and torsion groups control group underwent a sham operation, while in the second group the left testes were twisted clockwise and either testes in each group. Both testes in the torsion group had significantly more arterioles conby 720°. All rats underwent bilateral orchidectomy 24 h after the initial intervention. Three slides for taining blood cells than those in the control group. The diÂerence between the testes in the torsion group each testis (n=240) were evaluated randomly while unaware of treatment to determine the total number was also significant, but was not in the control group. Conclusion There was histological evidence of decreased of arterioles, the percentage of collapsed and open arterioles, the diameter of open arterioles and the blood flow in the contralateral testis during unilateral testicular torsion; contralateral testicular damage presence or absence of blood cells in the lumen. DiÂerences were assessed using t-tests for paired and during unilateral testicular torsion may result from hypoxia caused by decreased blood flow. independent samples. Results Very few arterioles were collapsed in both testes Keywords Spermatic cord torsion, testicular injury, contralateral of the control group and in the ipsilateral testes of the torsion group, but half the arterioles in the contralatepisodes of contralateral testicular torsion and underlying

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Quantitative evaluation of testicular biopsies from men with unilateral torsion of spermatic cord

Cited by 32 publications

References 18 publications

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

Hemodynamic Monitoring of the Contralateral Testis during Unilateral Testicular Torsion Describes the Mechanism of Damage

Histological evidence of decreased contralateral testicular blood flow during ipsilateral testicular torsion

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Quantitative evaluation of testicular biopsies from men with unilateral torsion of spermatic cord

Cited by 32 publications

References 18 publications

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24&nbsp;h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24&nbsp;h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

Hemodynamic Monitoring of the Contralateral Testis during Unilateral Testicular Torsion Describes the Mechanism of Damage

Histological evidence of decreased contralateral testicular blood flow during ipsilateral testicular torsion

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Product

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DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis

DNA Flow Cytometry Reveals Depressed Spermatogenetic Activity in the Contralateral Testis within 24 h of Ipsilateral Spermatic Cord Torsion Independently of the Presence of the Testis and Epididymis