“…Once initiated, dysmotility leads to reduced colonic transit, with decreased fecal output, increased dehydration of intestinal contents, dehydration of gastric contents with trichobezoar formation, impaired cecal fermentation, and disruption of the enteric microflora, creating a cycle of further anorexia and worsening stasis. 12,13 In severe cases, stasis leads to partial or complete obstruction or accumulation of gas within the GI tract (bloat) that can be a life-threatening emergency (discussed later). Clinical signs of GI stasis can include decreased or absent fecal material, anorexia, bruxism, pain with abdominal palpation, decreased GI sounds, dehydration, abdominal distension, gastric tympany, and respiratory or cardiovascular compromise.…”