Raeder syndrome associated with lesions of the internal carotid artery.

Healy, John F.; Zyroff, Jack; Rosenkrantz, Harvey

doi:10.1148/radiology.141.1.7291513

Cited by 9 publications

(5 citation statements)

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“…One case of internal carotid artery stenosis was excluded because the stenosis might have been due to dissection (Grimson case 12) (6). At least one case was excluded because the clinical features suggested the diagnosis of carotid artery dissection (7). Some cases reported as examples of Raeder's syndrome were excluded because they appeared to be better classified as cluster headache.…”

Section: Methodsmentioning

confidence: 99%

Benign Raeder's Syndrome is Probably a Manifestation of Carotid Artery Disease

Solomon

Lustig

2001

Cephalalgia

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Raeder first described Horner's syndrome with ipsilateral head pain due to paratrigeminal organic disease, but most subsequent reports of this syndrome were idiopathic. Our recent case prompted a review of past reports of idiopathic Raeder's syndrome. Because in recent years the features of Raeder's syndrome have been recognized as common manifestations of carotid artery dissection, we divided the cases into those with and those without carotid imaging studies. The classifications and differential diagnoses are discussed. Sixteen cases with carotid imaging studies were not very different from the 28 cases without such studies. Most studies were performed several weeks after onset of symptoms and carotid dissection could not be definitely excluded in any case. In most cases of idiopathic Raeder's syndrome, carotid artery dissection was not considered and in no case was that condition definitively excluded. People with Raeder's syndrome not associated with a paratrigeminal organic lesion probably have a disease of the carotid artery. Because of the different criteria and classifications of Raeder's syndrome it is best to relegate this eponym to history.

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Section: Methodsmentioning

confidence: 99%

Benign Raeder's Syndrome is Probably a Manifestation of Carotid Artery Disease

Solomon

Lustig

2001

Cephalalgia

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“…21 Other lesions of the internal carotid artery are known to be associated with partial Horner's syndrome. 22 Impairment of the vasa nervorum to the sympathetic fibers has been postulated when ptosis and miosis accompany carotid atherosclerosis and thrombosis, as well as other diseases of the carotid artery. [22][23][24] Swelling and perhaps inflammation of the arterial wall during CH may transiently block the vasa nervorum.…”

Section: Introductionmentioning

confidence: 99%

Cluster Headache and the Nervus Intermedius

Solomon¹

1986

Headache

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SYNOPSIS The nervus intermedius (NI) appears to be the main conduit of cluster headache (CH). Parasympatheticimpulses via the NI account for the associated symptoms of ipsilateral tearing and redness of the eye,congestion of the nose and rhinorrhea. Vasodilatory stimuli to the internal carotid artery may result inpartial Horner's syndrome and set off a cascade of events causing pain, similar to the changes in theexternal carotid arterial tree during migraine. The pain of CH may also be carried by afferent impulsesaccompanying motor fibers of the deep facial muscles.The central nervous system (CNS) mechanisms responsible for the circannual recurrence of CH may beexcitatory with parasympathetic impulses channeled through the NI. Conversely, inhibitory CNSmechanisms may be invoked to explain long remissions, if CH were to be due to hypersensitivity of the NIas caused by the subtle injury of a cross compressing blood vessel.

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“…8 In humans, involvement of the muscles and ligaments is traditionally accepted; and dissecting aneurysm of the cervical portion of the internal carotid artery is a well-documented complication of flexion-extension injuries. 9 There has been, however, scant attention given to the involvement of vagi or cervical sympathetic nerves. In the present study, vagi were spared and cervical sympathetic nerves were bilaterally affected.…”

Section: Methodsmentioning

confidence: 99%

Bilateral Sympathetic Dysfunction in Post‐Traumatic Headaches

Khurana

Nirankari²

1986

Headache

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SYNOPSIS Two patients developed generalized throbbing headaches, tenderness of the anterior neck and unilateral ptosis following whiplash injury. They were systematically evaluated for involvement of the central nervous system, carotid arteries, vagi and cervical sympathetic nerves. Biochemical study of pupils revealed bilateral postganglionic Horner's syndrome. Thermal‐induced facial sweating showed supraorbital anhidrosis. Follow‐up 18 to 54 months after injury demonstrated partial improvement in pupillary functions and headaches but persistent supraorbital anhidrosis. Pertinent literature on sympathetic dysfunction and post‐traumatic headaches is reviewed, and the reasons and implications for sympathetic involvement in hyperextension‐hyperflexion injury are discussed.

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Raeder syndrome associated with lesions of the internal carotid artery.

Cited by 9 publications

References 0 publications

Benign Raeder's Syndrome is Probably a Manifestation of Carotid Artery Disease

Benign Raeder's Syndrome is Probably a Manifestation of Carotid Artery Disease

Cluster Headache and the Nervus Intermedius

Bilateral Sympathetic Dysfunction in Post‐Traumatic Headaches

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