Recanalization of cerebral venous thrombosis within 24 hours

Abeele, B. Vanden; Saliou, Guillaume; Lehmann, P.; Théaudin, Marie; Kocheida, E.M.; Bonnaire, B.; Vallée, Jean-Noël

doi:10.1212/01.wnl.0000268500.32178.8d

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“…Indeed, successful recanalization has been reported with the use of intravenous UFH within the first 24 h of clinical presentation [9]. In situ endovascular thrombolysis is reserved for patients who have not responded to anticoagulation in the acute setting.…”

Section: Discussionmentioning

confidence: 99%

The “Cord Sign” in Cerebral Venous Thrombosis Associated with High Plasma Levels of Factor VIII

Giraldo

Petrinjac-Nenadic

2010

Neurocrit Care

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Section: Discussionmentioning

confidence: 99%

The “Cord Sign” in Cerebral Venous Thrombosis Associated with High Plasma Levels of Factor VIII

Giraldo

Petrinjac-Nenadic

2010

Neurocrit Care

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Early recanalisation of cerebral venous sinus thrombosis in an unusual case associated with severe protein S deficiency

Verma

Patil²,

Kumar³

2012

BMJ Case Reports

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An inherited or acquired deficiency of protein S leads to a prothrombotic state, with predisposition to venous thrombosis. We describe a case of cerebral venous sinus thrombosis (CVST) associated with acquired protein S deficiency and recanalisation within 15 days of anticoagulation. A 38-year-old man presented with recurrent headache, vomiting, altered sensorium and one episode of transient left hemiparesis. Magnetic resonance venography showed poor flow in the deep cerebral venous sinuses with extensive collateral venous channel formation, which resolved after 15 days of anticoagulation, along with clinical improvement. Serum protein S activity was found to be markedly low (16% of biological reference). CVST should be suspected in a patient with acute features of raised intracranial pressure or focal neurological deficit, and a patient without obvious clinical predisposition for a prothrombotic state should be evaluated for underlying thrombophilic states like protein S deficiency.

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Recanalization of cerebral venous thrombosis within 24 hours

Cited by 2 publications

References 3 publications

The “Cord Sign” in Cerebral Venous Thrombosis Associated with High Plasma Levels of Factor VIII

The “Cord Sign” in Cerebral Venous Thrombosis Associated with High Plasma Levels of Factor VIII

Early recanalisation of cerebral venous sinus thrombosis in an unusual case associated with severe protein S deficiency

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