2007
DOI: 10.1016/j.molimm.2006.12.010
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Recognition of H-2Kb by Ly49Q suggests a role for class Ia MHC regulation of plasmacytoid dendritic cell function

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Cited by 23 publications
(31 citation statements)
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“…As previously reported, L929 tumor cells are unable to stimulate lacZ production by BWZ. Ly49Q BALB or BWZ.Ly49Q B6 reporter cells (23). However, stable expression of H-2K b , but not H-2D b , in L929 was able to stimulate both types of BWZ.Ly49Q reporter cells, but not parental BWZ.36 parental cells, in line with previous findings (Fig.…”
Section: Resultssupporting
confidence: 91%
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“…As previously reported, L929 tumor cells are unable to stimulate lacZ production by BWZ. Ly49Q BALB or BWZ.Ly49Q B6 reporter cells (23). However, stable expression of H-2K b , but not H-2D b , in L929 was able to stimulate both types of BWZ.Ly49Q reporter cells, but not parental BWZ.36 parental cells, in line with previous findings (Fig.…”
Section: Resultssupporting
confidence: 91%
“…Binding was observed for Ly49G: (22). Similarly, using lacZ-based reporter cell assays, as well as soluble MHC-I tetramers, it was previously reported that the plasmacytoid dendritic cell (pDC)-expressed Ly49Q displays strong H-2K b binding, but apparently no binding to H-2D b or MHC-I alleles expressed in cells from mice possessing H-2 a,q,k,d MHC haplotypes (23). However, since these initial studies there have been new advances in our knowledge of Ly49/MHC-I interactions in the B6 inbred mouse strain, including the role of cis versus trans competitive binding and loss of binding observed when the whole MHC-I tetramer is not mouse derived (24).…”
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confidence: 60%
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