2020
DOI: 10.1111/cea.13570
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Relationship between immune parameters during a severe exacerbation in allergic asthmatic children and asthma outcomes in the following year

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Cited by 3 publications
(2 citation statements)
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“…Whereas viral infection did not modulate PRR expression and function on PBMC as compared with non-infected asthmatic patients, a reduced expression of PRR was reported in a sub-group of children prone to reinfection outside an exacerbation (steady state 8 weeks later), and displaying neutrophilic airway inflammation away from exacerbation [10,49]. Although present both at exacerbation and steady state, this impairment of PRR expression as well as the inflammatory profile was not associated with the clinical evolution and risk of exacerbations in the year following exacerbation [50].…”
Section: Virus Recognition During Asthma Attack Is Mainly Driven By Airway Epithelial Cellsmentioning
confidence: 89%
“…Whereas viral infection did not modulate PRR expression and function on PBMC as compared with non-infected asthmatic patients, a reduced expression of PRR was reported in a sub-group of children prone to reinfection outside an exacerbation (steady state 8 weeks later), and displaying neutrophilic airway inflammation away from exacerbation [10,49]. Although present both at exacerbation and steady state, this impairment of PRR expression as well as the inflammatory profile was not associated with the clinical evolution and risk of exacerbations in the year following exacerbation [50].…”
Section: Virus Recognition During Asthma Attack Is Mainly Driven By Airway Epithelial Cellsmentioning
confidence: 89%
“…We have finally entered the much‐anticipated era of personalized medicine for asthma, and several articles evaluated biologics and the variation in individual response to these in people with asthma 25‐27 , 28‐36 , 37 , 38 Others evaluated more traditional treatments for asthma, such as corticosteroids or mechanisms that may underlie impaired airway function in asthma 39‐59 . New interventions studied in clinical trials included air cleaners, triple inhaler therapy, an oral prostaglandin D2 antagonist 60‐62 and other more preliminary data from various interventions, from cinnamon and bacterial peptides to allergen immunotherapy 63‐67 .…”
Section: Asthmamentioning
confidence: 99%