“…Although low density lipoprotein (LDL) has been most thoroughly investigated, studies have shown that like oxidized LDL, oxidized intermediate density lipoprotein (1DL) leads to increased cholesterol uptake by macro-, phages [12] and to endothelial vasoraotor dysfunction [13]. Thus, it is increasingly clear that triglyceride rich proteins, especially DDL and" other -very low density lipoprotein remnants are also atherogenic [14,15].…”