Renal and Bladder Function Recovery after Prostatectomy in
Patients with a Chronic Residual Urine of above 1,000 ml

Nissenkorn, I; Savion, M.; Servadio, C

doi:10.1159/000473002

Cited by 14 publications

(8 citation statements)

References 3 publications

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“…In the current retrospective analysis of 2741 men presenting with LUTS due to BPH, the observed incidences of CKD, as defined by elevated serum creatinine levels, were similar to data reported by others [10,11]. However, a wide range of renal failure rate among patients with BPH has so far been reported, which might be due to inconsistent methods of defining renal dysfunction [12][13][14][15][16][17]. Among the present patients with LUTS from BPH, Q max and a history of DM and/or hypertension were independent predictors of the presence of CKD as defined by serum creatinine level or eGFR.…”

Section: Discussionsupporting

confidence: 87%

Chronic kidney disease among men with lower urinary tract symptoms due to benign prostatic hyperplasia

et al. 2010

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Section: Discussionsupporting

confidence: 87%

Chronic kidney disease among men with lower urinary tract symptoms due to benign prostatic hyperplasia

et al. 2010

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“…One study found a 7.7% prevalence of renal failure in men presenting for prostate surgery compared to a 3.7% prevalence in age-matched men presenting for nonprostate surgery [24]. Many studies of men with renal failure and bladder outlet obstruction show improvement in renal function in the follow-up after prostate surgery [25][26][27]. Case reports have also shown dialysis-dependent patients recover renal function up to a year after having prostate surgery [28].…”

Section: Discussionmentioning

confidence: 99%

The association between benign prostatic hyperplasia and chronic kidney disease in community-dwelling men

Rule¹,

Jacobson²,

Roberts³

et al. 2005

Kidney International

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“…Although upper tract dilatation in BOO is an indicator of the need for surgical intervention, its sensitivity is known to be low. 1,22 Several parameters including the prostate or transitional zone volume, PVR volume and urinary flow rate can be obtained using non-invasive diagnostic testing. However, they have limited value for the aetiological evaluation or definite diagnosis of BOO; instead, they only raise suspicion of BOO.…”

Section: Discussionmentioning

confidence: 99%

Perirenal fat stranding on CT: is there an association with bladder outlet obstruction?

Han¹,

Sung²,

Kim³

et al. 2016

BJR

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Objective: To determine the association between perirenal fat stranding (PFS) on CT and bladder outlet obstruction (BOO). Methods: CT scans from 122 patients who had undergone urodynamic study for lower urinary tract symptoms (LUTS) were registered after exclusion of patients with renal or retroperitoneal disease. Images were independently reviewed by two radiologists and compared with those of 244 age-and sex-matched control patients without LUTS. The PFS severity was scored on a fourpoint scale, and the interobserver agreement was assessed with kappa statistics. The severity score and incidence was compared between the groups, and the association with baseline characteristics was analyzed. For the LUTS group, an association between PFS severity and urodynamic and laboratory data was evaluated.Results: PFS was more frequent and more severe in the LUTS group than in the control group (p-value , 0.001); its presence was significantly associated with male gender and older age (p-value , 0.001). PFS was predominantly bilateral in both groups (80.1-93.2%). In the LUTS group, PFS severity scores were significantly correlated with the maximum flow rate, maximum detrusor pressure and estimated glomerular filtration rate (p-value , 0.001). Interobserver agreements were excellent for PFS presence (k 5 0.883) and severity (k 5 0.816). Conclusion: Severe PFS was observed in older, male patients with LUTS. PFS severity was associated with the degree of BOO and impaired renal function. Advances in knowledge: Recognition of PFS on the CT scan may warrant further evaluation of BOO and appropriate management to prevent renal impairment. INTRODUCTIONBladder outlet obstruction (BOO) is a common cause of lower urinary tract symptoms (LUTS) in males, and benign prostatic hyperplasia (BPH) is mainly attributable to BOO in male patients. Multiple mechanisms leading to increased pressure in the urinary bladder and the renal collecting system have been proposed to explain the association between chronic BOO and renal function impairment.1 Renal function impairment secondary to BOO is preventable if cases are recognized early, but it is often difficult to identify patients at risk of renal failure. Urodynamic study is the gold standard for the diagnosis of BOO.2 However, because of its invasiveness, its use for screening purposes is inappropriate.

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Renal and Bladder Function Recovery after Prostatectomy in Patients with a Chronic Residual Urine of above 1,000 ml

Cited by 14 publications

References 3 publications

Chronic kidney disease among men with lower urinary tract symptoms due to benign prostatic hyperplasia

Chronic kidney disease among men with lower urinary tract symptoms due to benign prostatic hyperplasia

The association between benign prostatic hyperplasia and chronic kidney disease in community-dwelling men

Perirenal fat stranding on CT: is there an association with bladder outlet obstruction?

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