“…The pathogenesis of endometriosis is still unclear. Several hypotheses have been established to explain the development of endometriosis, including retrograde menstruation and ectopic transplantation, coelomic metaplasia, iatrogenic injury, autoimmunity, embryonic theory and stem cell origin, genetic predisposition, hormones and others [ 3 , 9 , 18 , 19 ]. An increased level of Interleukin-1, Interleukin-2, tumor necrosis factor and prostaglandins have been found in the endometrial lesions [ 3 ].…”