Resistance to extinction in an odor–20% CO2 inhalation paradigm: Further evidence for a symptom learning account of multiple chemical sensitivity

Multiple chemical sensitivity is a response to chemicals acting as toxicants via excessive NMDA activity

“…[2,3,12]). What do Meulders et al [1] have to say about this extensive literature? Nothing; they pretend it does not exist.…”

“…Meulders et al [1] claim to have presented evidence for symptom learning in multiple chemical sensitivity (MCS). They make this claim even though they studied only normal people and they studied the responses of such normals to chemicals not implicated in MCS.…”

“…Thus, we have compelling evidence that MCS is a response to chemicals acting as toxicants, with these chemicals acting to produce a common response that is otherwise implicated in MCS. What do Meulders et al [1] have to say about this extensive evidence that chemicals in MCS act as toxicants in the body? Nothing; they pretend that none of this literature exists.…”

“…These studies tell us that chemicals act as toxicants in MCS and that genes that influence their metabolism to either less or more active compounds influence, therefore, susceptibility to MCS. What do Meulders et al [1] have to say about these compelling genetic studies of MCS? Nothing; they pretend they do not exist.…”

“…[2]). What do Meulders et al [1] have to say about the animal model studies of MCS? Nothing, they write as if these studies do not exist.…”