Respiratory dysfunction in thrombotic thrombocytopenic purpura

Bone, Roger C.; Henry, Joseph E.; Petterson, Jay; Amare, Mammo

doi:10.1016/0002-9343(78)90818-5

Cited by 61 publications

(40 citation statements)

References 47 publications

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“…[27][28][29][30][31][32][33][34][35][36][37][38][39][40][41][42] Among these 16 patients, 13 were men, opposite to the gender disparity of TTP-HUS 25 but consistent with the greater frequency of acute pancreatitis among men. 43 The etiologies of the pancreatitis in these 16 patients were typical for acute pancreatitis among adults: 43,44 seven cases were related to chronic severe alcohol use, five to gallbladder disease, and four were idiopathic.…”

Section: Previously Reported Patientsmentioning

confidence: 96%

“…ADAMTS13 activity was reported only for patient 15; her activity was slightly low (53%, normal range 67-177%) without a demonstrable inhibitor. 41 Ten patients were treated with plasma exchange, four with plasma infusion, one with splenectomy, 27 and one with only supportive care including hemodialysis. 32 Using the criteria in Table 1, there was a possible relation between the pancreatitis and the subsequent development of TTP-HUS in eight patients (Table 3).…”

Section: Previously Reported Patientsmentioning

confidence: 99%

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Pancreatitis preceding acute episodes of thrombotic thrombocytopenic purpura-hemolytic uremic syndrome: report of five patients with a systematic review of published reports

Swisher¹,

Doan²,

Vesely³

et al. 2007

Haematologica

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Section: Previously Reported Patientsmentioning

confidence: 96%

Section: Previously Reported Patientsmentioning

confidence: 99%

Pancreatitis preceding acute episodes of thrombotic thrombocytopenic purpura-hemolytic uremic syndrome: report of five patients with a systematic review of published reports

Swisher¹,

Doan²,

Vesely³

et al. 2007

Haematologica

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“…Du r a t i o n f r o m S y mp t o m On s e t t o Di a g n o s i s o f I n t r a p u l mo n a r y He mo r r h a g e , T r e a t me n t s a n d Ou t c o me syndrome, hardly being differentiated from each other, was reported occasionally (15,16). Goodpasture syndrome or positive GBM has been reported in several CVD cases (17)(18)(19)(20) (1,3,(21)(22)(23)(24)(25). Recently, in contrast to that deficient ADAMTS13 activity causes idiopathic or hereditary TTP, endothelial involvement has been shown to contribute to TTP pathogenesis in CVD patients (26,27 …”

Section: Discussionmentioning

confidence: 99%

Intrapulmonary Hemorrhage in Collagen-Vascular Diseases Includes a Spectrum of Underlying Conditions

Kobayashi

Inokuma

2009

Intern. Med.

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“…21 Myocardial failure and non-bacterial thrombotic endocarditis have also been described. [22][23][24] The reasons for the high incidence of TM observed in our study are unclear. None of the cytotoxics used in our 535 Table 2 Outcome after phases III to V See text for case report.…”

Section: Discussionmentioning

confidence: 99%

Thrombotic microangiopathy: a new dose-limiting toxicity of high-dose sequential chemotherapy

Vantelon

Bourhis

et al. 2001

Bone Marrow Transplant

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Summary:Ten patients with refractory (n ‫؍‬ 8) or early relapsing (n ‫؍‬ 2) aggressive non-Hodgkin's lymphoma were enrolled in a pilot study evaluating a high-dose sequential chemotherapy regimen with peripheral blood stem cell (PBSC) support. Five treatment phases were scheduled: phase I (cyclophosphamide ؉ etoposide followed by lenograstim (G-CSF), and a PBSC harvest); phase II (cisplatinum ؉ cytarabine ؉ etoposide followed by lenograstim); phases III and IV (cyclophosphamide ؉ cytarabine ؉ etoposide followed by autologous PBSC infusion and lenograstim); and phase V (carmustine ؉ cytarabine ؉ etoposide ؉ melphalan followed by autologous PBSC infusion and lenograstim). Ten, nine, eight, six and four of the 10 patients received one, two, three, four and five of the five scheduled phases of treatment, respectively. Four patients were withdrawn from the study due to progressive disease and two due to thrombotic microangiopathy (TM). Moreover, in the four patients who completed all treatment phases, an additional case of TM was seen. In all three patients with TM, laboratory studies showed evidence of Coombs negative hemolytic anemia, thrombocytopenia, renal dysfunction and in addition cardiac failure in two patients. TM may be a new dose-limiting toxicity of high-dose sequential chemotherapy followed by repeated PBSC transplantation. Bone Marrow Transplantation (2001) 27, 531-536. Keywords: thrombotic microangiopathy; lymphoma; chemotherapy The role of high-dose chemotherapy (HDC) with autologous stem cell support in relapsed non-Hodgkin's lymphoma (NHL) has been clearly demonstrated in patients with chemosensitive relapse. 1,2 However, NHL patients who do not achieve a first complete remission or experience early relapse still have a very poor prognosis even when HDC

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Respiratory dysfunction in thrombotic thrombocytopenic purpura

Cited by 61 publications

References 47 publications

Pancreatitis preceding acute episodes of thrombotic thrombocytopenic purpura-hemolytic uremic syndrome: report of five patients with a systematic review of published reports

Pancreatitis preceding acute episodes of thrombotic thrombocytopenic purpura-hemolytic uremic syndrome: report of five patients with a systematic review of published reports

Intrapulmonary Hemorrhage in Collagen-Vascular Diseases Includes a Spectrum of Underlying Conditions

Thrombotic microangiopathy: a new dose-limiting toxicity of high-dose sequential chemotherapy

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