2002
DOI: 10.1006/clim.2002.5214
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Respiratory Reovirus 1/L Induction of Diffuse Alveolar Damage: Pulmonary Fibrosis Is Not Modulated by Corticosteroids in Acute Respiratory Distress Syndrome in Mice

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Cited by 21 publications
(37 citation statements)
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“…26 Thus, we analyzed cytokines and chemokines that have previously been associated with fibrotic lesion development. We believe that our data supports a role for IFN-␥ in the fibrotic process and that it acts as a profibrotic agent in the spectrum of fibrosis induced in reovirus 1/L-infected mice.…”
Section: Discussionmentioning
confidence: 99%
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“…26 Thus, we analyzed cytokines and chemokines that have previously been associated with fibrotic lesion development. We believe that our data supports a role for IFN-␥ in the fibrotic process and that it acts as a profibrotic agent in the spectrum of fibrosis induced in reovirus 1/L-infected mice.…”
Section: Discussionmentioning
confidence: 99%
“…19 -21 We have described a spectrum of inflammatory lung diseases after respiratory infection with reovirus serotype 1, strain Lang (reovirus 1/L), which is dependent on the strain of mice used. [22][23][24][25][26][27] In this spectrum CBA/J mice infected with 1 ϫ 10 6 PFU reovirus 1/L develop a histologically severe inflammation characterized by an infiltration of lymphocytes organized adjacent to the pulmonary vasculature of the lung. 23 This pattern of mononuclear cell organization without the involvement of intraluminal fibrosis results in lesions histopathologically consistent with the non-fibrotic human syndrome termed follicular bronchiolitis (FB).…”
Section: Bronchiolitis Obliterans Organizing Pneumonia (Boop) Ismentioning
confidence: 99%
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