RESTORATION OF RETINAL BLOOD FLOW VIA TRANSLUMENAL Nd:YAG EMBOLYSIS/EMBOLECTOMY (TYL/E) FOR CENTRAL AND BRANCH RETINAL ARTERY OCCLUSION

Opremcak, E. Mitchel; Rehmar, Alan J.; Ridenour, Chet D.; Borkowski, Lisa M.; Kelley, James K.

doi:10.1097/iae.0b013e31814b1d6e

Cited by 62 publications

(44 citation statements)

References 12 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Framme et al [47] found no statistically significant visual improvement in CRAO patients treated with thrombolytic therapy when compared with those treated with other common treatments. Some experts have concluded that despite its efficacy in treating acute ischemic stroke, conflating the management of CRAO with that of acute ischemic stroke is erroneous [23,66]. This notion is underscored by the finding that over 80% of emboli in CRAO consist of cholesterol or calcified material, which are not theoretically treatable with thrombolytic therapy [23].…”

Section: Future Prospectsmentioning

confidence: 93%

Thrombolytic therapy in central retinal artery occlusion: cutting edge therapy, standard of care therapy, or impractical therapy?

Hazin

Dixon²,

Bhatti³

2009

Current Opinion in Ophthalmology

View full text Add to dashboard Cite

show abstract

Section: Future Prospectsmentioning

confidence: 93%

Thrombolytic therapy in central retinal artery occlusion: cutting edge therapy, standard of care therapy, or impractical therapy?

Hazin

Dixon²,

Bhatti³

2009

Current Opinion in Ophthalmology

View full text Add to dashboard Cite

show abstract

“…For example, it has been claimed that YAG laser embolysis/embolectomy in BRAO resulted in VA improvement (Mason et al, 2007; Opremcak et al, 2008). As discussed elsewhere (Hayreh, 2008), there were flaws in the study by Opremcak et al (2008) which invalidate their claims; moreover, the procedure resulted in complications in 47%, including vitreous/subhyaloid hemorrhages.…”

Section: Natural History Of Visual Outcome In Branch Retinal Artementioning

confidence: 99%

Ocular vascular occlusive disorders: Natural history of visual outcome

Hayreh

2014

Progress in Retinal and Eye Research

271

197

View full text Add to dashboard Cite

Ocular vascular occlusive disorders collectively constitute the most common cause of visual disability. Before a disease can be managed, it is essential to understand its natural history, so as to be able to assess the likely effectiveness of any intervention. I investigated natural history of visual outcome in prospective studies of 386 eyes with non-arteritic anterior ischemic optic neuropathy (NA-AION), 16 eyes with non-arteritic posterior ischemic optic neuropathy, 697 eyes with central retinal vein occlusion (CRVO), 67 eyes with hemi-CRVO (HCRVO), 216 eyes with branch retinal vein occlusion (BRVO), 260 eyes with central retinal artery occlusion (CRAO), 151 eyes with branch retinal artery occlusion (BRAO) and 61 eyes with cilioretinal artery occlusion (CLRAO). My studies have shown that every one of these disorders consists of multiple distinct clinical sub-categories with different visual findings. When an ocular vascular occlusive disorder is caused by giant cell arteritis, which is an ophthalmic emergency, it would be unethical to do a natural history study of visual outcome in them, because in this case early diagnosis and immediate, intensive high-dose steroid therapy is essential to prevent any further visual loss, not only in the involved eye but also in the fellow, normal eye. In NA-AION in eyes seen ≤2 weeks after the onset, visual acuity (VA) improved in 41% of those with VA 20/70 or worse, and visual field (VF) improved in 26% of those with moderate to severe VF defect. In non-ischemic CRVO eyes with VA 20/70 or worse, VA improved in 47% and in ischemic CRVO in 23%; moderate to severe VF defect improved in 79% in non-ischemic CRVO and in 27% in ischemic CRVO. In HCRVO, overall findings demonstrated that initial VA and VF defect and the final visual outcome were different in non-ischemic from ischemic HCRVO – much better in the former than the latter. In major BRVO, in eyes with initial VA of 20/70 or worse, VA improved in 69%, and moderate to severe VF defect improved in 52%. In macular BRVO with 20/70 or worse initial VA, it improved in 53%, and initial minimal-mild VF defect was stable or improved in 85%. In various types of CRAO there are significant differences in both initial and final VA and VF defects. In CRAO eyes seen within 7 days of onset and initial VA of counting fingers or worse, VA improved in 82% with transient non-arteritic CRAO, 67% with non-arteritic CRAO with cilioretinal artery sparing, 22% with non-arteritic CRAO. Central VF improved in 39% of transient non-arteritic CRAO, 25% of non-arteritic CRAO with cilioretinal artery sparing and 21% of non-arteritic CRAO. Peripheral VF improved in non-arteritic CRAO in 39% and in transient non-arteritic CRAO in 39%. In transient CRAO, finally peripheral VFs were normal in 93%. In non-arteritic CRAO eyes initially 22% had normal peripheral VF and in the rest it improved in 39%. Final VA of 20/40 or better was seen in 89% of permanent BRAO, and in 100% of transient BRAO and non-arteritic CLRAO. In permanent BRAO eyes, among those seen wit...

show abstract

“…Other treatments reported with variable success include isovolemic hemodilution; hyperbaric oxygen; intravenous steroid to reduce vascular endothelial; Nd:YAG laser arteriotomy and embolectomy [6]; surgical embolectomy [7]; and intraarterial fibrinolysis delivered directly into the ophthalmic artery [8]. However, none of these methods are proven to be effective.…”

Section: Discussionmentioning

confidence: 99%

Why can’t I see after my heart is fixed: a case series of ocular complications after cardiac intervention

et al. 2016

View full text Add to dashboard Cite

BackgroundThe purpose of this study was to report case series of retinal artery occlusion (RAO) as one of the significant complication post cardiac intervention.Case presentationWe are reporting one case of branch RAO and one case of central RAO after percutaneous coronary intervention (PCI). In case 1, a 74-year-old gentleman with pre-existing diabetes mellitus and hypertension was electively admitted for PCI for coronary artery disease (CAD) in our centre (UKMMC). Few hours after the procedure, patient complained of sudden blurring of vision in the right eye. He was found to have branch retinal artery occlusion. In case 2, a 49-year-old gentleman presented with ST segment elevation myocardial infarction (STEMI) and had an emergency PCI performed 2 h upon admission. He noticed sudden dropped of vision in his right eye immediately after the procedure. He was diagnosed to have central retinal artery occlusion.ConclusionsIn conclusion, retinal artery occlusion is a possible complication post PCI. Patients need to be informed especially in high risk cases.

show abstract

RESTORATION OF RETINAL BLOOD FLOW VIA TRANSLUMENAL Nd:YAG EMBOLYSIS/EMBOLECTOMY (TYL/E) FOR CENTRAL AND BRANCH RETINAL ARTERY OCCLUSION

Cited by 62 publications

References 12 publications

Thrombolytic therapy in central retinal artery occlusion: cutting edge therapy, standard of care therapy, or impractical therapy?

Thrombolytic therapy in central retinal artery occlusion: cutting edge therapy, standard of care therapy, or impractical therapy?

Ocular vascular occlusive disorders: Natural history of visual outcome

Why can’t I see after my heart is fixed: a case series of ocular complications after cardiac intervention

Contact Info

Product

Resources

About