Reversible catecholamine-induced cardiomyopathy due to pheochromocytoma: Case report

Methamphetamine‐related cardiovascular diseases

“…For one, the constantly elevated level of catecholamines seems to play a major role in the pathogenesis of MA‐associated DCM. In agreement with this pathology, cases of catecholamine‐induced DCM due to pheochromocytoma are reported, where the LVEF recovered after operative treatment . Also, the histological features of MA‐associated cardiomyopathy (MACM) and catecholamine‐induced DCM have been shown to be similar …”

Acute myocardial infarction as the first manifestation of paraganglioma

“…15 Several complications have been described associated with functional PGLs, including LV hypertrophy, ischemic heart disease, myocardial infarction, cardiac arrhythmias, Takotsubo cardiomyopathy, dilated cardiomyopathy, and shock. 16 In this case, the PGL can explain both events: AMI of the inferior wall, due to the above reasons, and recrudescence of chest pain associated with elevation of MNb and dynamic electrocardiographic changes, by coronary spasm due to the paroxysmal release of catecholamines. Nevertheless, it is important to mention, while the etiology was not clarified, that the patient was treated with a β-blocker (bisoprolol) due the ACS.…”

Pheochromocytoma, “the Great Masquerader,” Presenting as Severe Acute Decompensated Heart Failure in a Young Patient

“…Its annual incidence is approximately 0.8 per 100,000 person-years and probably occurs in less than 0.5 percent of patients with hypertension [ 1 , 2 ]. Pheochromocytoma can be sporadic or familial; sporadic forms are usually diagnosed between the ages of 40 and 50, while the hereditary forms present in childhood or early adulthood [ 3 ]. This tumor has varied clinical manifestations ranging from an asymptomatic subclinical picture to symptoms such as headaches, palpitations, sweating, anxiety, emesis, hypertension, and, in rare cases, multiorgan failure [ 4 – 7 ].…”

“…Pheochromocytomas have been associated with other various cardiovascular complications due to overwhelming catecholamine levels. The most common complications include left ventricular hypertrophy, ischemic heart disease, disturbances in rhythm and conduction, malignant ventricular arrhythmias, and even shock [ 3 , 5 , 11 ]. The potentially fatal cardiovascular complications of these tumors are due to the potent effects of catecholamines, especially noradrenaline, the main transmitter released from sympathetic nerve terminals.…”

“…The classical clinical triad is described with headaches, palpitations, and sweating. However, this tumor can be asymptomatic or present with a myriad of symptoms including headaches, anxiety attacks, hypertension, and in rare cases, multiorgan failure [ 1 – 3 ]. These symptoms are secondary to excess catecholamines.…”