Right Auricular and Ventricular Pressure Patterns in Constrictive Pericarditis

Yu, Paul N.; Lovejoy, Frank W.; Joos, Howard A.; Nye, Robert E.; Mahoney, Earl B.

doi:10.1161/01.cir.7.1.102

Cited by 57 publications

(11 citation statements)

References 3 publications

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“…Usually, the diagnosis is made on the basis of cardiac catheterisation, as, in pericardial constriction, this will show equal or near equal diastolic pressures in both ventricles, without significant pulmonary hypertension 5. A recent analysis has shown that unless three criteria are all satisfied—that is, a difference of <5 mm Hg between directly measured end diastolic pressures in the two ventricles, a peak right ventricular pressure of less than 50 mm Hg, and a ratio of right ventricular end diastolic to peak pressure of more than 0.33, haemodynamic assessment is not discriminating (table3).…”

Section: Discussionmentioning

confidence: 99%

Restrictive pericarditis

Henein

Rakhit

Sheppard

et al. 1999

Heart

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Background-Pericardial thickening is an uncommon complication of cardiac surgery. Objectives-To study pericardial thickening as the cause of severe postoperative venous congestion. Subjects-Two men, one with severe aortic stenosis and single coronary artery disease, and one with coronary artery disease after an old inferior infarction. Both had coronary artery bypass grafting surgery. Methods-Magnetic resonance imaging (MRI), Doppler echocardiography, and cardiac catheterisation.Results-Venous pressure was raised in both patients. MRI showed mildly thickened pericardium, and cardiac catheterisation indicated diastolic equalisation of pressures in the four chambers. Jugular venous pulse showed a dominant "Y" descent coinciding with early diastolic flow in the superior vena cava, and mitral and tricuspid Doppler forward flow proved restrictive physiology. The clinical background suggested pericardial disease so both patients had pericardiectomy. This proved the pericardium to be thickened; the extent of fibrosis also involved the epicardium. Conclusions-Although rare, restrictive pericarditis (restrictive ventricular physiology resulting from pericardial disease) should be considered to be a separate diagnostic entity because its pathological basis and treatment are diVerent from intrinsic myocardial disease. This diagnosis may be confirmed by standard investigational techniques or may require diagnostic thoracotomy. (Heart 1999;82:389-392)

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Section: Discussionmentioning

confidence: 99%

Restrictive pericarditis

Henein

Rakhit

Sheppard

et al. 1999

Heart

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“…These measurements are shown in Table 1 and included RAP, pulmonary artery systolic pressure, right ventricular end-diastolic pressure (RVEDP), pulmonary capillary wedge pressure, left ventricular end-diastolic pressure (LVEDP), and height of the rapid filling wave (Table 1). Conventional hemodynamic criteria for the differentiation of CP from RMD have relied on the relationship between RVEDP and LVEDP, as well as the secondary effect on pulmonary pressures (4,(7)(8)(9), and are outlined in Table 2.…”

Section: Methodsmentioning

confidence: 99%

Constrictive Pericarditis in the Modern Era

Talreja

Nishimura

et al. 2008

Journal of the American College of Cardiology

216

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“…It is particularly useful where definite QRS, S-T -and T-wave abnormalities are present. Common errors encountered in this area are as follows: it is not generally appreciated that about 15 per cent of patients with myocardial infarction will demonstrate no electrocardiographic abnormalities. All too often only a single tracing is recorded, and it is not uncommon for electrocardiographic abnormalities to -appear several days after an episode of chest pain.…”

Section: Pericardial Diseasementioning

confidence: 99%