“…When rupture involves these sites they are often associated with an underlying pathology such as rheumatoid arthritic changes in the bone, pyogenic infections, gout, syphilis, tuberculosis, gonorrhoea, rough volar surface of the carpal bones, hook of hamate non-union, repetitive motions, prominent implants or degeneration of tendon substance associated with age. [2][3][4][5][6][7][8][9][10][11][12] The FDP avulsion is demonstrated when there is no active flexion at the DIP joint with stabilization of the proximal interphalangeal joint (PIP) joint. The additional signs reported are change in the normal cascade of the fingers, loss of DIP flexion on squeezing of the forearm muscle and loss of tenodesis in the affected finger.…”