Seizures and Encephalopathies following Metrizamide Myelography and Cisternography

Potts, Kevin M.

doi:10.1080/00029238.1990.11080319

Cited by 2 publications

(1 citation statement)

References 14 publications

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“…These include baclofen (Hormes et al 1988, Zak et al 1994, levodopa (Neufeld 1992), lithium (Smith and Kocen 1988), ifosamide (Simonian et al 1993, Wengs et al 1993, and metrizamide (Potts 1990). Some authors have felt that occasionally these patterns may represent nonconvulsive status epilepticus, while others view it as an encephalopathy that resolves with discontinuing the offending agent.…”

Section: Drug Intoxicationmentioning

confidence: 99%

EEG in Encephalopathy and Coma

Brenner

2003

American Journal of Electroneurodiagnostic Technology

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Despite advances in neuroimaging, the EEG remains extremely helpful in the evaluation of encephalopathy and coma. This review discusses a variety of encephalopathies and associated EEG findings. These include metabolic disturbances , such as hepatic or renal dysfunction, which are often characterized by slowing of background rhythms and triphasic waves. Hypoxia and drug intoxications can produce a number of abnormal EEG patterns such as burstsuppression, alpha coma, and spindle coma. Patients can be encephalopathic because of ongoing epileptic activity with minimal or no motor movements. This entity, nonconvulsive status epilepticus, is difficult to diagnose in obtunded!comatose patients. An EEG, which is required to verify the diagnosis, can also help monitor trreament.

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Section: Drug Intoxicationmentioning

confidence: 99%

EEG in Encephalopathy and Coma

Brenner

2003

American Journal of Electroneurodiagnostic Technology

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The Interpretation of the EEG in Stupor and Coma

Brenner

2005

The Neurologist

118

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This review discusses a variety of causes of stupor and coma and associated electroencephalographic (EEG) findings. These include metabolic disturbances such as hepatic or renal dysfunction, which are often characterized by slowing of background rhythms and triphasic waves. Hypoxia and drug intoxications can produce a number of abnormal EEG patterns such as burst suppression, alpha coma, and spindle coma. Structural lesions, either supra- or infratentorial, are reviewed. EEGs in the former may show focal disturbances such as delta and theta activity, epileptiform abnormalities, and attenuation of faster frequencies. In infratentorial lesions, the EEG may appear normal, particularly with a pontine lesion. Some patients may be encephalopathic because of ongoing epileptic activity with minimal or no motor movements. This entity, nonconvulsive status epilepticus (NCSE), is difficult to diagnose in obtunded/comatose patients, and an EEG is required to verify the diagnosis and to monitor treatment. Several EEG patterns and their interpretation in suspected cases of NCSE such as periodic lateralized epileptiform discharges (PLEDs), bilateral independent periodic lateralized epileptiform discharges (BIPLEDs), generalized periodic epileptiform discharges (GPEDs), and triphasic waves are reviewed. Other entities discussed include the locked-in syndrome, neocortical death, persistent vegetative state, brainstem death, and brain death.

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Seizures and Encephalopathies following Metrizamide Myelography and Cisternography

Cited by 2 publications

References 14 publications

EEG in Encephalopathy and Coma

EEG in Encephalopathy and Coma

The Interpretation of the EEG in Stupor and Coma

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