Seizures in activating calcium‐sensing receptor antibodies positive autoimmune hypoparathyroidism and preponderance of steroids therapy: A case report

Khan, Qaisar Ali; Shahi, Ameena; Shahi, Alishba; Baqi, Abdul; Tango, Tamara; Verma, Ravina; Hamid, Asrah Abdul; Saravanan, Priyadharshini; Alam, Mazhar; Shinwari, Ismail Khan; Ain, Hoor Ul

doi:10.1002/ccr3.6901

Cited by 1 publication

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“…The desired therapeutic result is to maintain the serum calcium close to the lower limit of the normal range. 16 Evidence from prior case reports has suggested that correcting underlying abnormalities may improve the presenting symptoms in cases where metabolic disturbances are found. Since hypoparathyroidism is a common cause, we can set dietary regimens for the patient.…”

Section: Discussionmentioning

confidence: 99%

Fahr’s Syndrome Secondary to Primary Hypoparathyroidism Presenting With Seizures and the Role of Steroid Therapy

Shah,

Hadi,

Idrees

et al. 2023

Clin Med Insights Case Rep

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Background: Fahr’s syndrome a rare neurological condition characterized by an abnormal basal ganglia calcification. The condition has both genetic and metabolic causes. Here, we describe a patient who had Fahr’s syndrome and basal secondary to hypoparathyroidism, and her calcium level raised after the administration of steroid therapy. Case report: We presented a case of a 23-year-old female with seizures. Associated symptoms included headache, vertigo, disturbed sleep, and reduced appetite. Her laboratory workup revealed hypocalcemia and low parathyroid hormone level, computed tomographic (CT) scan of the brain showed diffuse calcification in the brain parenchyma. The patient was diagnosed as a case of Fahr’s syndrome secondary to hypoparathyroidism. The patient was started on calcium and calcium supplementations along with anti-seizure therapy. Her calcium level raised after the initiation of oral prednisolone and she remained asymptomatic. Conclusion: Steroid could be considered as an adjunct therapy with calcium and vitamin D supplementation in patient whose Fahr’s syndrome is secondary to primary hypoparathyroidism.

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Section: Discussionmentioning

confidence: 99%