Self-reported emotional eaters consume more food under stress if they experience heightened stress reactivity and emotional relief from stress upon eating

Klatzkin, Rebecca R.; Nolan, Laurence J.; Kissileff, Harry R.

doi:10.1016/j.physbeh.2021.113638

Cited by 23 publications

(11 citation statements)

References 70 publications

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“…Higher levels of emotional eating were associated with an increase in BMI over time ( β = 0.18, p = 0.004), according to a study conducted on a large adult population in the Netherlands ( n = 1562) [ 48 ]. A plausible explanation accounting for the observed association is that individuals may engage in emotional eating as an adaptive response to mitigate the impact of stress or negative emotional states, thereby potentially contributing to subsequent weight gain [ 49 , 50 ]. For example, emotional eating partially mediated the association between stress and body fat percentage in U.S. military personnel [ 20 ] indicating that stress or negative emotions may lead to a cycle of overeating as individuals attempt to cope with or suppress their negative feelings through eating [ 51 ].…”

Section: Discussionmentioning

confidence: 99%

Emotional Eating and Cardiovascular Risk Factors in the Police Force: The Carolina Blue Project

Wu,

Pacchioni,

Gehi

et al. 2024

IJERPH

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There is an association between emotional eating and cardiovascular disease (CVD) risk factors; however, little is known about this association in the police force. This study explores the associations between emotional eating and CVD risk factors in law enforcement officers in North Carolina. Four hundred and five officers completed The Emotional Eating Scale, and 221 of them completed the assessment for CVD-related markers. Descriptive statistics, Pearson’s correlation, and multiple linear regression analyses were performed. Emotional eating in response to anger was significantly positively associated with body weight (β = 1.51, t = 2.07, p = 0.04), diastolic blood pressure (β = 0.83, t = 2.18, p = 0.03), and mean arterial pressure (β = 0.84, t = 2.19, p = 0.03) after adjusting for age and use of blood pressure medicine. Emotional eating in response to depression was significantly positively associated with triglycerides (β = 5.28, t = 2.49, p = 0.02), while the emotional eating in response to anxiety was significantly negatively associated with triglycerides (β = −11.42, t = −2.64, p = 0.01), after adjusting for age and use of cholesterol medicine. Our findings offer new insights to address emotional eating and lower CVD risk in law enforcement officers.

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Section: Discussionmentioning

confidence: 99%

Emotional Eating and Cardiovascular Risk Factors in the Police Force: The Carolina Blue Project

Wu,

Pacchioni,

Gehi

et al. 2024

IJERPH

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“…The reward is not so much what people gain (the moti-vation to strive for something), but what people are running away from (motivation to escape trouble). Taking this into consideration and calling this phenomenon as seeking relief helps people struggling with eating disorders by: 1) Noticing that people seek relief not debauchery or hedonism, 2) Craving for reducing tension may lead to design other ways of reducing tension and replace BED/OCD behaviors with them 3) Notice that the pursuit of relief is as strong as the pursuit of pleasure, Self-reported emotional eaters consume more food under stress if they experience heightened stress reactivity and emotional relief from stress upon eating [21]. People who are emotional eaters are more likely to feel out of control and stressed.…”

Section: Strategies For Stress Mitigation -Need For Emotion Of Reliefmentioning

confidence: 99%

The Role of Emotional Eating as Relief Mechanism from Psychological Distress and its Impact on Overall Wellbeing

Jurek,

Maruda

2024

Epidemiol Public Health

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Antibiotic susceptibility profiles of bacterial isolates from immunocompromised patients attending selected health facilities in Benue State were investigated. Three hundred and three midstream urine samples were collected for both study and control patient and bacteria were isolated and identified following standard bacteriological methods. Prevalence of UTIs and antibiotic susceptibility profiles were done. Drug resistant bacterial isolates were identified as multidrug resistant (MDR), extensive drug resistant (XDR) and pan-drug resistant organisms (PDR). Data were analysed and statistical significance was considered at p<0.05. From the results, fifty-seven and twenty-one bacterial isolates were identified given prevalence of 18.8% and 6.9% for the study and control subjects respectively. The male HIV patients had higher prevalence of UTI (20.6%) than the female (17.5%). The study bacterial isolates expressed high levels of resistance to ampicillin, aztreonam, nalidixic acid, tetracycline, cotrimoxazole and the quinolones. Cefotaxime, genticin, augmentin and imipenem which showed high susceptibility were the most effective antibiotics. None of the isolates showed complete susceptibility to all the tested antibiotics. Forty-one drug resistant bacterial isolates consisting of 30 MDR, 7 XDR and 4 PDR isolates were identified. High prevalence of UTI and drug resistant isolates in immunocompromised HIV infected patients was shown in this study. Therefore, regular test of urine cultures and sensitivity, antibiotic policy for rational use of antibiotics in our hospitals and close monitoring of MDR, XDR and PDR isolates in our communities will significantly reduce the problem of antibiotic resistance.

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“…Immediately after a stressful event, there is a CRH-mediated suppression of food intake, followed by a CRH/ACTH-dependent, glucocorticoid mediated, stimulation of hunger and eating behavior ( 37 ). Accordingly, chronic stress, and the resultant chronic activation of the hypothalamic-pituitary adrenal axis, induces weight gain in rodents and humans where it is known as “emotional eating” or “stress-induced eating” ( 38 – 41 ). In addition to the hyperglycemic effects of AVP described above, it is likely that AVP produces behavioral changes via V 1 b potentiation of the CRF-ACTH-glucocorticoid axis.…”

Section: Arginine Vasopressin Fructose and The Metabolic Syndromementioning

confidence: 99%

THIRSTY FOR FRUCTOSE: Arginine Vasopressin, Fructose, and the Pathogenesis of Metabolic and Renal Disease

Student

Sowers

Lockette

2022

Front. Cardiovasc. Med.

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We review the pathways by which arginine vasopressin (AVP) and hydration influence the sequelae of the metabolic syndrome induced by high fructose consumption. AVP and inadequate hydration have been shown to worsen the severity of two phenotypes associated with metabolic syndrome induced by high fructose intake–enhanced lipogenesis and insulin resistance. These findings have implications for those who frequently consume sweeteners such as high fructose corn syrup (HFCS). Patients with metabolic syndrome are at higher risk for microalbuminuria and/or chronic kidney disease; however, it is difficult to discriminate the detrimental renal effects of the metabolic syndrome from those of hypertension, impaired glucose metabolism, and obesity. It is not surprising the prevalence of chronic renal insufficiency is growing hand in hand with obesity, insulin resistance, and metabolic syndrome in those who consume large amounts of fructose. Higher AVP levels and low hydration status worsen the renal insufficiency found in patients with metabolic syndrome. This inter-relationship has public health consequences, especially among underserved populations who perform physical labor in environments that place them at risk for dehydration. MesoAmerican endemic nephropathy is a type of chronic kidney disease highly prevalent in hot ambient climates from southwest Mexico through Latin America. There is growing evidence that this public health crisis is being spurred by greater fructose consumption in the face of dehydration and increased dehydration-dependent vasopressin secretion. Work is needed at unraveling the mechanism(s) by which fructose consumption and increased AVP levels can worsen the renal disease associated with components of the metabolic syndrome.

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Self-reported emotional eaters consume more food under stress if they experience heightened stress reactivity and emotional relief from stress upon eating

Cited by 23 publications

References 70 publications

Emotional Eating and Cardiovascular Risk Factors in the Police Force: The Carolina Blue Project

Emotional Eating and Cardiovascular Risk Factors in the Police Force: The Carolina Blue Project

The Role of Emotional Eating as Relief Mechanism from Psychological Distress and its Impact on Overall Wellbeing

THIRSTY FOR FRUCTOSE: Arginine Vasopressin, Fructose, and the Pathogenesis of Metabolic and Renal Disease

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