Sensory form of acute polyneuritis

Dawson, David M.; Samuels, Martin A.; Morris, James

doi:10.1212/wnl.38.11.1728

Cited by 41 publications

(21 citation statements)

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“…Cerebrospinal fluid analysis and electrophysiological tests are compatible with Guillain-Barré syndrome 8889 A necropsy study showed demyelination, with mononuclear cell infiltration of nerves and posterior roots 89…”

Section: Clinicopathological Typesmentioning

confidence: 95%

Guillain-Barré syndrome

Seneviratne

2000

Postgraduate Medical Journal

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Guillain-Barré syndrome is an autoimmune disorder encompassing a heterogeneous group of pathological and clinical entities. Antecedent infections are thought to trigger an immune response, which subsequently cross reacts with nerves leading to demyelination or axonal degeneration. Both intravenous immunoglobulin treatment and plasma exchange have been found to be equally beneficial. Several factors are useful in predicting the outcome of these patients. (Postgrad Med J 2000;76:774-782)

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Section: Clinicopathological Typesmentioning

confidence: 95%

Guillain-Barré syndrome

Seneviratne

2000

Postgraduate Medical Journal

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“…In others, evidence of demyelination was confined to the terminal latencies, as observed in previously reported cases. [9][10][11] This is understandable, given the nature of the clinical presentation of sensory GBS. The difficulty in demonstrating demyelination in sensory nerve conduction is due to the technical limitation of sensory NCS performed with surface electrodes.…”

Section: (M In 2; All In 6)mentioning

confidence: 99%

“…10 At autopsy, there was extensively lymphocytic infiltration of nerves and posterior roots, with sparing of the anterior roots. Teased fiber preparation of nerve showed a demyelinating lesion.…”

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confidence: 99%

Sensory Guillain–Barré syndrome

LaGanke²,

Claussen³

2001

Neurology

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Article abstract-Objective: To report eight cases of sensory Guillain-Barré syndrome (GBS). Background: The concept of sensory equivalent to ascending paralysis of GBS was raised in 1958, and the diagnostic criteria for a sensory loss and areflexia variant of GBS were proposed in 1981. However, clinical cases meeting these criteria have been relatively scarce. Methods: During a 13-year period between 1986 and 1999, the authors collected eight cases of an acute sensory demyelinating neuropathy that met most of the proposed diagnostic criteria of a sensory variant of GBS. Results: In all patients, sensory neuropathy was sudden at onset and peaked to maximal deficit within 4 weeks. In five (63%) cases, there was an antecedent viral illness. All patients had objective sensory loss and diminished or absent reflexes. None showed any muscle weakness. In all four patients in whom the spinal fluid was examined during the first 4 weeks, there was albuminocytologic dissociation. All of the patients had electrophysiologic evidence of demyelination in at least two nerves. Demyelination was demonstrated in motor nerve conduction in seven patients and in sensory nerve conduction in one, indicating that motor nerve conduction studies were the key for the diagnosis of demyelinating neuropathy. All patients had sensory nerve conduction abnormalities in at least one nerve. Three patients responded to immunotherapies. All had a favorable outcome, with a monophasic course of disease and no sign of relapse. Conclusion: The current study confirms the existence of sensory GBS.

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“…The six patients had a good prognosis, considering the effects of the small fiber damage. Dawson et al (8) described a case of sensory GBS in which the patient exhibited abnormal sensation and joint position sense, vibratory sensory abnormalities and ataxia. Certain patients may exhibit subjective weak limb muscle strength, and electrophysiology tests can show demyelination of the involved motor fiber, which is also considered as a lesion in the large sensory fiber.…”

Section: Discussionmentioning

confidence: 99%

Sensory Guillain-Barré syndrome: A case report

Zhang

Liu

Zhang

et al. 2014

Experimental and Therapeutic Medicine

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A 58-year-old female exhibited the onset of symmetrical sensory abnormalities of the face and extremities. The neurological examination revealed normal muscle strength with abated or absent tendon reflexes. The patient experienced symmetrical glove- and stocking-type pinprick sensations in the distal extremities and a loss of temperature sensation, but had normal proprioception and vibration senses and joint topesthesia. The lumbar puncture showed protein cell separation at the fifth week after the onset of symptoms. At the same time-point, the electrophysiological examination showed demyelination changes involving the trigeminal nerve and the somatic motor nerve. Needle electromyography revealed normal results. The clinical symptoms ceased progression at the fourth week after symptom onset, and began to improve from the sixth. This case was considered to be sensory Guillain-Barré syndrome, which was characterized by its cranial nerve involvement.

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Sensory form of acute polyneuritis

Cited by 41 publications

References 0 publications

Guillain-Barré syndrome

Guillain-Barré syndrome

Sensory Guillain–Barré syndrome

Sensory Guillain-Barré syndrome: A case report

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