Serological evidence of human adenovirus infections in animals

Shortridge, K. F.; Hu, Leyong; A, Lansdell

doi:10.1016/0021-9975(76)90072-4

Cited by 4 publications

(2 citation statements)

References 14 publications

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“…The day 1093 NOT spectra, the INT spectrum and the UKIRT spectra were reduced using the standard routines of the FIGARO package (Shortridge 2002). The other NOT spectra were reduced using the standard procedures within IRAF.…”

Section: Spectroscopymentioning

confidence: 99%

On the source of the late-time infrared luminosity of SN 1998S and other Type II supernovae

Pozzo¹,

Meikle²,

Fassia³

et al. 2004

Monthly Notices of the Royal Astronomical Society

150

209

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We present late‐time near‐infrared (NIR) and optical observations of the Type IIn SN 1998S. The NIR photometry spans 333–1242 d after explosion, while the NIR and optical spectra cover 333–1191 and 305–1093 d, respectively. The NIR photometry extends to the M′ band (4.7 μm), making SN 1998S only the second ever supernova for which such a long IR wavelength has been detected. The shape and evolution of the Hα and He i 1.083‐μm line profiles indicate a powerful interaction with a progenitor wind, as well as providing evidence of dust condensation within the ejecta. The latest optical spectrum suggests that the wind had been flowing for at least 430 yr. The intensity and rise of the HK continuum towards longer wavelengths together with the relatively bright L′ and M′ magnitudes show that the NIR emission was due to hot dust newly formed in the ejecta and/or pre‐existing dust in the progenitor circumstellar medium (CSM). The NIR spectral energy distribution (SED) at about 1 yr is well described by a single‐temperature blackbody spectrum at about 1200 K. The temperature declines over subsequent epochs. After ∼2 yr, the blackbody matches are less successful, probably indicating an increasing range of temperatures in the emission regions. Fits to the SEDs achieved with blackbodies weighted with λ−1 or λ−2 emissivity are almost always less successful. Possible origins for the NIR emission are considered. Significant radioactive heating of ejecta dust is ruled out, as is shock/X‐ray‐precursor heating of CSM dust. More plausible sources are (a) an IR echo from CSM dust driven by the ultraviolet/optical peak luminosity, and (b) emission from newly‐condensed dust which formed within a cool, dense shell produced by the ejecta shock/CSM interaction. We argue that the evidence favours the condensing dust hypothesis, although an IR echo is not ruled out. Within the condensing‐dust scenario, the IR luminosity indicates the presence of at least 10−3 M⊙ of dust in the ejecta, and probably considerably more. Finally, we show that the late‐time (K–L′)0 evolution of Type II supernovae may provide a useful tool for determining the presence or absence of a massive CSM around their progenitor stars.

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Section: Spectroscopymentioning

confidence: 99%

On the source of the late-time infrared luminosity of SN 1998S and other Type II supernovae

Pozzo¹,

Meikle²,

Fassia³

et al. 2004

Monthly Notices of the Royal Astronomical Society

150

209

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show abstract

“…y inhibitors are active against human influenza A viruses of the H2 and H3 subtypes but not the H1 subtype (8,9). 'y inhibitors are present at high titer in horse and guinea pig sera and have been identified as a2-macroglobulins.…”

mentioning

confidence: 99%

Bovine and mouse serum beta inhibitors of influenza A viruses are mannose-binding lectins.

Anders

Hartley

Jackson

1990

Proc. Natl. Acad. Sci. U.S.A.

180

175

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Normal bovine and mouse sera contain a component, termed ( inhibitor, that . The association of (3 inhibitor resistance with loss of this carbohydrate side chain suggested that (3 inhibitors may be lectins. In support of this hypothesis, treatment of the 13 inhibitorsensitive parent virus Mem71H-BelN with periodate converted it to the resistant state. Furthermore, the inhibitory activity of both bovine and mouse sera for the parental virus was abrogated by D-mannose. We conclude that the 13 inhibitors in bovine and mouse sera are mannose-binding lectins that inhibit hemagglutination and neutralize virus infectivity by binding to carbohydrate at the tip of the HA spike, blocking access of cell-surface receptors to the receptor-binding site on HA.

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Incomplete human adenovirus replication in canine tumour cells and serological evidence of infections in tumour-bearing canine pets

Winters

Young

1980

Journal of Comparative Pathology

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Serological evidence of human adenovirus infections in animals

Cited by 4 publications

References 14 publications

On the source of the late-time infrared luminosity of SN 1998S and other Type II supernovae

On the source of the late-time infrared luminosity of SN 1998S and other Type II supernovae

Bovine and mouse serum beta inhibitors of influenza A viruses are mannose-binding lectins.

Incomplete human adenovirus replication in canine tumour cells and serological evidence of infections in tumour-bearing canine pets

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