Sir: A 70-year-old man was admitted to our emergency center with left thalamic hemorrhage and, because of his comatose state and hypoxemia, he was managed with a respirator in the intensive care unit. On the 11th day of admission, his neurological status had improved and oxygenation was good without mechanical ventilation and, therefore, the endotracheal tube was removed. Since he could not yet eliminate sputa sufficiently by himself, percutaneous minitracheotomy (Mini-Trach II, Portex) was chosen to extract them. It was performed with no problem using the standard technique [1], and could help the sputum suction. However, 4 days after the minitracheotomy had been performed the patient's respiratory rate suddenly increased from 20 to 35/min with mild inspiratory stridor and, at the same time, oxygen saturation decreased to 90 % despite oxygen inhalation. We soon opened the minitracheotomy tube and supplied oxygen through it, and the tachypnea, desaturation and inspiratory stridor disappeared immediately. Emergency bronchoscopy was performed at once, showing that a granuloma,