Silent Thyroiditis following Rubella.

Nakamura, Shigenori; Kosaka, J; Sugimoto, Miyuki; Shima, Hiroto; Takuno, Hiroshi

doi:10.1507/endocrj1954.37.79

Cited by 10 publications

(10 citation statements)

References 20 publications

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“…In 1990, de Bruin et al [12] reported the presence of atypical subacute thyroiditis, which developed in 12 patients, including two family members in the same period (July-September). In both reports, flulike symptoms and liver dysfunction were observed in most of the patients, suggesting that viral infection might be a cause of thyrotoxicosis [8][9][10]. However, no evidence of a viral infection was obtained in these studies.…”

Section: Discussionmentioning

confidence: 82%

“…Thus, it is suggested that iodine intake and various cytokines can initiate silent thyroiditis [3]. In addition, silent thyroiditis can also develop in patients after the cessation of steroid treatment for rheumatoid arthritis [4], after adrenalectomy in patients with Cushing's syndrome [5,6], after the administration of gonadotropin-releasing hormone analogues [7], and after viral infections [8][9][10]. However, the outbreak of silent thyroiditis (or atypical thyroiditis) has been indicated in few reports [11,12] Here, we report the cases of two brothers and their mother who developed silent thyroiditis in spring.…”

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confidence: 99%

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Familial Occurrence of Silent Thyroiditis

2005

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Abstract. Two brothers and their mother, who had allergic rhinitis caused by Japanese cedar pollen, developed silent thyroiditis in spring. In addition, these three patients had the same HLA haplotype. Another brother, the father, and the paternal grandmother, who did not have allergic rhinitis caused by Japanese cedar pollen, did not show symptoms of silent thyroiditis. The present study indicates that genetic and/or environmental factors are important in the development of this familial type of silent thyroiditis.

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Section: Discussionmentioning

confidence: 82%

mentioning

confidence: 99%

Familial Occurrence of Silent Thyroiditis

2005

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“…An environmental toxic insult has been proposed in endocrine autoimmune processes as a trigger event for the disease in genetically susceptible individuals (Toni010 et al,, 1980;Nakamura et al,, 1990;Toussaint-Demylle et al, 1990;Sundick et al, 1992;Cavallo et al, 1992). How may this affect adhesion molecules?…”

Section: Lymphocyte Inflltratlon and Types Of Adheslon In Autoimmune mentioning

confidence: 99%

Lymphocytic traffic and homing into target tissue and the generation of endocrine autoimmunity

Pozzilli

Carotenuto

Delitala

1994

Clinical Endocrinology

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Summary Endocrine autoimmunity is known to be characterized by the presence of specific autoantibodies and from the histopathological point of view by lymphocytic inflitration in the target tissue. The presence of mononuclear cell infiltrates is the pathological hallmark of most endocrine diseases characterized by an autoimmune process directed against antigens expressed on endocrine cells. Infiltrating cells can usually be detected by biopsy or by using other, non‐invasive, techniques. However, in endocrine tissue such as the islets of Langerhans and the adrenal glands it is difficult to perform biopsies and diagnosis of the autoimmune process is dependent mainly upon detection of specific autoantibodies. A crucial aspect of endocrine autoimmunity and of all processes of organ specific autoimmunity is why and how lymphocytes migrate from primary lymphoid tissue to their specific targets. This occurs mainly through contact with specific adhesion molecules which enable lymphocytes to adhere to the endothelial vessels. In close proximity to the target tissue. In this review we discuss the homing of peripheral mononuclear cells into target endocrine tissues and the mediating role of adhesion molecules.

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“…Die Patienten haben keine lokalen Beschwerden, insbesondere keine Schmerzen. Auffallend diskrepant sind häufig Höhe der Schilddrüsenhormonkonzentrationen und geringe bis fehlende klinische Beschwerden einer Hyperthyreose[12,32,33]. der Sonografie Das sonografische Bild ist variabel.…”

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Die Bedeutung der Sonografie bei der Abklärung von Schilddrüsenerkrankungen

Dietrich

Bojunga

2015

Z Gastroenterol

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Thyroid nodules and thyroid abnormalities are common findings in the general population. Ultrasonography is the most important imaging tool for diagnosing thyroid disease. In the majority of cases a correct diagnosis can already be made in synopsis of the sonographic together with clinical findings and basal thyroid hormone parameters and an appropriate therapy can be initiated thereafter. A differentiation of hormonally active versus inactive nodes, and in particular benign versus malignant nodules is sonographically, however, not reliably possible. In this context, radioscanning has its clinical significance predominantly in diagnosing hormonal activity of thyroid nodules. Efforts of the past years aimed to improve sonographic risk stratification to predict malignancy of thyroid nodules through standardized diagnostic assessment of evaluated risk factors in order to select patients, who need further diagnostic work up. According to the "Breast Imaging Reporting and Data System" (BI-RADS), "Thyroid Imaging Reporting and Data Systems" (TI-RADS) giving standardized categories with rates of malignancy were evaluated as a basis for further clinical management. Recent technological developments, such as elastography, also show promising data and could gain entrance into clinical practice. The ultrasound-guided fine-needle aspiration is the key element in the diagnosis of sonographically suspicious thyroid nodules and significantly contributes to the diagnosis of malignancy versus benignity.

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Silent Thyroiditis following Rubella.

Cited by 10 publications

References 20 publications

Familial Occurrence of Silent Thyroiditis

Familial Occurrence of Silent Thyroiditis

Lymphocytic traffic and homing into target tissue and the generation of endocrine autoimmunity

Die Bedeutung der Sonografie bei der Abklärung von Schilddrüsenerkrankungen

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