Síndrome de disfunción autonómica secundario a meningitis tuberculosa

Antón, J. Gil; Bayón, Jeremías; Fernández, Y. López; Pilar, Javier

doi:10.1016/s1695-4033(04)78427-1

Cited by 5 publications

(2 citation statements)

References 7 publications

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“…We also found occurrence of PSH in one case of tuberculous meningitis and in one case of JE; this was an interesting finding as PSH had been very rarely reported in association with tuberculous meningitis previously. [ 11 ] We could not find any previous report of the occurrence of PSH following JE.…”

Section: Discussionmentioning

confidence: 94%

Paroxysmal sympathetic hyperactivity in neurological critical care

Verma¹,

Giri²,

Rizvi³

2015

Indian Journal of Critical Care Medicine

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Introduction:Paroxysmal sympathetic hyperactivity (PSH) is a clinical disorder mainly caused by traumatic brain injury, stroke, encephalitis and other types of brain injury. The clinical features are episodes of hypertension, tachycardia, tachypnea, fever and dystonic postures. In this study, we described clinical profile and outcome of six patients of PSH admitted in neurocritical care unit.Materials and Methods:This was a prospective observational study conducted at neurology critical care unit of a tertiary care center. All patients admitted at neurology critical unit during 6-month period from August 2013 to January 2014 were screened for the occurrence of PSH. The clinical details and outcome was documented.Results:PSH was observed in 6 patients. Male to female ratio was 5:1. Mean age ± SD was 36.67 ± 15.19 years. The leading causes were traumatic brain injury (two patients), stroke (two patients) and Japanese encephalitis (JE) (one patient) and tuberculous meningitis (one patient).Conclusion:PSH is an unusual complication in neurocritical care. It prolonged the hospitalization and hampers recovery. The other life-threatening conditions that mimic PSH should be excluded. The association with JE and tuberculous meningitis was not previously described in literature.

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Section: Discussionmentioning

confidence: 94%

Paroxysmal sympathetic hyperactivity in neurological critical care

Verma¹,

Giri²,

Rizvi³

2015

Indian Journal of Critical Care Medicine

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“…It is rarely encountered in patients with CNS infections. Two individual case reports of patients with tuberculous meningitis manifesting symptoms of PAID are described in the literature [3, 4]. Attacks of agitation, hypertension, fever, autonomic dysfunction, and extensor posturing are highly suggestive of PAID syndrome.…”

Section: Introductionmentioning

confidence: 99%

Paroxysmal Autonomic Instability with Dystonia after Pneumococcal Meningoencephalitis

Safadieh

Sharara‐Chami

Dabbagh

2012

Case Reports in Medicine

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Streptococcus pneumoniae is a common cause of bacterial meningitis, frequently resulting in severe neurological impairment. A seven-month-old child presenting with Streptococcus pneumoniae meningoencephalitis developed right basal ganglia and hypothalamic infarctions. Daily episodes of agitation, hypertension, tachycardia, diaphoresis, hyperthermia, and decerebrate posturing were observed. The diagnosis of paroxysmal autonomic instability with dystonia was established. The patient responded to clonidine, baclofen, and benzodiazepines. Although this entity has been reported in association with traumatic brain injury, and as a sequel to some nervous system infections, this is the first case, to our knowledge, associated with pneumococcal meningoencephalitis.

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Paroxysmal autonomic instability with dystonia in a patient with tuberculous meningitis: a case report

Ramdhani¹,

Sikma

Witkamp

et al. 2010

J Med Case Reports

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IntroductionThis case report describes an extremely rare combination of paroxysmal autonomic instability with dystonia and tuberculous meningitis. Paroxysmal autonomic instability with dystonia is normally associated with severe traumatic brain injury.Case presentationA 69-year-old man of Indonesian descent was initially suspected of having a community-acquired pneumonia, which was seen on chest X-ray and computed tomography of the chest. However, a bronchoscopy showed no abnormalities. He was treated with amoxicillin-clavulanic acid in combination with ciprofloxacin. However, nine days after admission he was disorientated and complained of headache. Neurological examination revealed no further abnormalities. A lumbar puncture revealed no evidence of meningitis. He was then transferred to our hospital. At that time, initial cultures of bronchial fluid for Mycobacterium tuberculosis turned positive, as well as polymerase chain reaction for Mycobacterium tuberculosis. Later, during his stay in our intensive care unit, he developed periods with hypertension, sinus tachycardia, excessive transpiration, decreased oxygen saturation with tachypnea, pink foamy sputum, and high fever. This constellation of symptoms was accompanied by dystonia in the first days. These episodes lasted approximately 30 minutes and improved after administration of morphine, benzodiazepines or clonidine. Magnetic resonance imaging showed an abnormal signal in the region of the hippocampus, thalamus and the anterior parts of the lentiform nucleus and caudate nucleus.ConclusionsIn patients with (tuberculous) meningitis and episodes of extreme hypertension and fever, paroxysmal autonomic instability with dystonia should be considered.

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Síndrome de disfunción autonómica secundario a meningitis tuberculosa

Cited by 5 publications

References 7 publications

Paroxysmal sympathetic hyperactivity in neurological critical care

Paroxysmal sympathetic hyperactivity in neurological critical care

Paroxysmal Autonomic Instability with Dystonia after Pneumococcal Meningoencephalitis

Paroxysmal autonomic instability with dystonia in a patient with tuberculous meningitis: a case report

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