Síndrome do Cabrito Mole ("Floppy Kid")

Riet-Correa, Franklin; Tabosa, Ivon M.; Vasconcelos, Jackson S. de; Medeiros, Josemar Marinho de

doi:10.1590/s0100-736x2004000200011

Cited by 12 publications

(4 citation statements)

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“…7 The mortality rate is usually 30-50%. 2 One of the criteria for inclusion of ill animals in the present study was metabolic acidosis without dehydration. 1,4 The anion gap, which is the difference between strong cations and strong anions, was significantly higher in affected animals than in control animals.…”

Section: Discussionmentioning

confidence: 99%

Floppy Kid Syndrome Caused by D‐Lactic Acidosis in Goat Kids

Bleul

Schwantag

Stocker

et al. 2006

Veterinary Internal Medicne

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Section: Discussionmentioning

confidence: 99%

Floppy Kid Syndrome Caused by D‐Lactic Acidosis in Goat Kids

Bleul

Schwantag

Stocker

et al. 2006

Veterinary Internal Medicne

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“…In the case of sudden death, the diagnosis of enzootic ataxia can be made by assessing the liver or serum levels of the mineral; however, the assessment of deaths on the property is especially important to rule out poisoning by toxic plants (Riet-Correa et al 2001). It is noteworthy that, in the present study, a differential diagnosis was made with plant poisoning with neurological changes and sudden death, cerebellar hypoplasia, meningitis, intestinal parasitism (Radostitis et al 2007), soft goat syndrome (Riet-Correa et al 2004), caprine arthritis encephalitis (Riet-Correa et al 2001), polioencephalomalacia (Lima et al 2005) and oropharyngeal and neurological lesions associated with the use of metering guns (Sant' Ana et al 2007). For such differentiation, we performed pasture inspections, clinical and epidemiological data evaluation, macroscopic and histopathological examinations, as well as copper dosages.…”

Section: Clinical Aspectsmentioning

confidence: 72%

“…No citations of prophylactic treatments of this nature were found in the literature. Such treatment also avoids the intoxication of sheep as they should not consume mineral mixtures with more than 0.4%-0.8% copper sulfate (Riet-Correa et al 2004). However, for proper prophylaxis, soil correction/fertilization and supplementation of pregnant females is recommended to prevent the occurrence of the congenital form of the disease (Tokarnia et al 1971, Peixoto et al 2005.…”

Section: Treatmentmentioning

confidence: 99%

Outbreak of enzootic ataxia in goats and sheep in the state of Bahia

et al. 2019

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The objective of this study was to describe for the first time the clinical-epidemiological, laboratory and pathological aspects of different forms of enzootic ataxia in goats and lambs in the state of Bahia, as well as to propose an emergency oral treatment with copper sulfate in an individualized way for neonates. Three outbreaks of enzootic ataxia were studied. The history, clinical signs and epidemiological data were obtained from the owners and checked during the visits. The first outbreak (FO) occurred in 2013 in a property located in the municipality of Itaberaba and affected a herd composed of 90 goats and 130 sheep. The second outbreak (SO) occurred in 2014 in the municipality of Santa Luz and affected a herd of 90 goats and 110 sheep. The third outbreak (TO) occurred in 2018, in a property located in the same municipality of the SO, being the herd constituted by 80 sheep. Samples of blood were collected from all goats and sheep treated for serum copper dosage. Nine animals (five goats and four sheep) that presented a more severe clinical picture and unfavorable prognosis were necropsied for diagnostic confirmation. During the necropsy of the FO, SO and TO animals, liver samples were collected for copper dosage as well as fragments of several organs for histopathology. At the site where the SO occurred, samples of soil and the main forages where the animals were kept were collected to determine copper, iron, molybdenum, sulfur and zinc contents. The clinical and histopathological findings in the three outbreaks studied were characteristic of enzootic ataxia. In the outbreaks studied, the frequency of enzootic ataxia was higher in goats (52) than in lambs (39). In the goats the mean values of serum copper (0.05mg/kg) and hepatic (2.48mg/kg) of the FO and SO were well below their respective reference values, 12 times lower than serum levels and eight times lower in relation to liver contents. On the other hand, TO sheep presented mean values of serum copper (0.015mg/kg) reduced by 40 times. The levels of iron and sulfur were high in the fodder of the property where the SO occurred, already in the soil, iron levels were high and copper levels were reduced. The treatment tested in neonates and lambs with two doses of copper sulphate solution orally at a dose of 20mg/kg for sheep and 35mg/kg for goats during the first and second week of life was effective in prevention of enzootic ataxia in neonates and can be used for emergency control of the disease. The occurrence of enzootic ataxia (congenital and late form) in goats and sheep was first reported in Bahia. It is also worth noting that this disease caused by copper deficiency has caused great damage to the breeders of small ruminants, mainly due to the high mortality of the animals affected.

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“…A variety of predisposing factors and causes of FKD has been proposed . An effect of breed and specific disease‐causing agents such as rotaviruses, enteropathic Escherichia coli , Campylobacter jejuni , Cryptosporidium parvum , Giardia intestinalis, and Clostridium perfringens type D could not be established .…”

Section: Discussionmentioning

confidence: 99%

Quantitative Analysis of Fecal Flora in Goat Kids with and without Floppy Kid Syndrome

Bleul¹,

Fassbind

Ghielmetti³

et al. 2013

Veterinary Internal Medicne

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Background: Floppy kid syndrome (FKS) affects goat kids in the first month of life and is associated with high morbidity and mortality rates. The condition is characterized by neurological signs that can be ascribed to increased plasma D-lactate concentrations. The source of D-lactate has not been identified conclusively, but D-lactate-producing bacteria in the large intestine are thought to be involved.Objectives: To determine the number of colony-forming unit (CFUs) of certain groups of bacteria in the feces of kids with and without FKS.Animals: Nineteen goat kids with clinical signs of FKS, acidemia (pH ≤ 7.2), and plasma D-lactate concentration >7 mM and 15 healthy goat kids without acidemia (pH >7.2) and D-lactate concentration <1 mM.Methods: In this case-control study, the goat kids were examined clinically and blood was collected to measure D-lactate concentration, blood gases, and acid-base parameters. Fecal samples were collected and the total aerobic bacterial count and CFU counts of coliforms, enterococci, staphylococci, streptococci, lactobacilli, and clostridia were determined using the surface plating method.Results: Goat kids with FKS had a mean plasma D-lactate concentration of 10.9 AE 3.7 mM compared with 0.3 AE 0.9 mM in healthy kids, and significantly greater CFU counts for enterococci, streptococci, staphylococci, and lactobacilli than healthy kids.Conclusions and Clinical Importance: The groups of bacteria present in greater numbers in the feces of goat kids with FKS include several D-lactate-producing species, which makes dysbacteriosis a likely cause of the increased plasma D-lactate concentration in FKS.

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Síndrome do Cabrito Mole ("Floppy Kid")

Cited by 12 publications

References 0 publications

Floppy Kid Syndrome Caused by D‐Lactic Acidosis in Goat Kids

Floppy Kid Syndrome Caused by D‐Lactic Acidosis in Goat Kids

Outbreak of enzootic ataxia in goats and sheep in the state of Bahia

Quantitative Analysis of Fecal Flora in Goat Kids with and without Floppy Kid Syndrome

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