Sodium and water depletion with acute malignant hypertension

Barraclough, M. A.

doi:10.1016/0002-9343(66)90107-0

Cited by 73 publications

(21 citation statements)

References 32 publications

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“…MV hring et al (1975a, b, 1976), Barraclough (1966), and Kramer et al (1974) emphasized that the presence of renal artery stenosis is essential for the onset of malignant hypertension in rats and that the excess sodium and water excretion is a secondary phenomenon. In our case, however, renal artery stenosis was not recognized either macroscopically or histologically.…”

Section: Histological Findings O F the Kidneysmentioning

confidence: 99%

“…Dialysis treatment was started on September 2,1975. From November 1975 massive amounts of sodium (5,000 mEq or more monthly) and water (26 liters or more monthly) were removed by the dialysis.…”

mentioning

confidence: 99%

“…The Ministry of Health and Welfare, Japan recommended the following criteria for the diagnosis of malignant hypertension in 1971; Group A malignant hypertension -(1) diastolic hypertension constantly higher than 130 mmHg, (2) papilledema, (3) advanced renal damage, and (4) poor general conditions accompanied by heart failure or hypertensive cerebrovascular symptoms. Group B malignant hypertension -three of the 4 items mentioned above are positive, and the remaining one falls a little bit short of the criterion.…”

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confidence: 99%

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Development of malignant hypertension in patients with uremia under hemodialysis: A case report and discussions on its etiology.

Kuroda

Murata

Maeta

et al. 1981

Tohoku J. Exp. Med.

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Section: Histological Findings O F the Kidneysmentioning

confidence: 99%

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confidence: 99%

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confidence: 99%

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Development of malignant hypertension in patients with uremia under hemodialysis: A case report and discussions on its etiology.

Kuroda

Murata

Maeta

et al. 1981

Tohoku J. Exp. Med.

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“…Barraclough (1966) also reported in man with right renal artery stenosis that acute progressive rise in blood pressure and excess renin production were responsible for the renal sodium and water wasting.…”

mentioning

confidence: 92%

“…To sum up, both Mohring et al (1975a, b) and Barraclough (1966) emphasized that the exsistence of unilateral renal artery stenosis is essential for the onset of malignant hypertension, regarding the excess sodium and water excretion as a secondary phenomenon.…”

mentioning

confidence: 99%

Treatment of malignant hypertension with infusion of sodium chloride; A case report and a review.

Kuroda

Yamauchi

Murata

et al. 1980

Tohoku J. Exp. Med.

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Severe Hypertension in Identical Twin Infants

Bergstein¹

1971

Arch Pediatr Adolesc Med

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A set of identical female twins developed hematuria and a salt-losing state three days after birth. Evaluation at 10 months of age for failure to thrive revealed severe hypertension associated with interstitial nephritis and massive medial hypertrophy of the renal arterioles. Known causes of hypertension have been eliminated. To our knowledge, this clinical picture has not been previously described.Hypertensi on in the pediatrie age group is an unusual and challenging problem. By careful clin¬ ical investigation and the use of new diagnostic techniques, hyper¬ tension in most children may be as¬ cribed to one of the more than 25 causes listed by Rubin1 and Loggie.2 Occasionally, as in adults,3 the cause of hypertension in childhood remains unexplained. Recent reports4·5 de¬ scribing children with unexplained or "essential" hypertension have charMinneapolis 55455 (Dr. Bergstein). acterized these patients as being asymptomatic with diastolic blood pressures less than 120 mm Hg, as having a strong family history of hy¬ pertensive disease, and as having a benign course. This report documents the in¬ vestigation of identical female twins with a destructive form of hyper¬ tension occurring in infancy which, by its early onset, severity, absence of definable cause, and lack of family history falls clearly outside previously described categories. Accordingly, this type of hypertensive disease is presented as a new entity. Report of CasesIdentical female infants were born to a 29-year-old white woman following a fullterm gestation complicated only by a third trimester pedal edema. The twins' sub¬ sequent course was remarkably similar.Case l.-The first-born developed gross hematuria on day 3. Results of physical ex¬ amination were normal and a flush blood pressure was 60 mm Hg. She was trans¬ ferred to Milwaukee Children's Hospital where a hemogram, prothrombin time, and platelet count were normal. Her urine con¬ tained protein (4 + ) and was still grossly bloody. A urine culture was sterile and the blood urea nitrogen (BUN) level was 41 mg/100 ml. The kidneys were not visual¬ ized by intravenous pyelogram and no source of bleeding was found at cystoscopy. A retrograde pyelogram and a Downloaded From: http://archpedi.jamanetwork.com/ by a University of Calgary User on 05/27/2015

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Sodium and water depletion with acute malignant hypertension

Cited by 73 publications

References 32 publications

Development of malignant hypertension in patients with uremia under hemodialysis: A case report and discussions on its etiology.

Development of malignant hypertension in patients with uremia under hemodialysis: A case report and discussions on its etiology.

Treatment of malignant hypertension with infusion of sodium chloride; A case report and a review.

Severe Hypertension in Identical Twin Infants

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