“…To keep labeled subjects with these minimal anomalies make them being periodically checked. Grade 2 VMs with dilated and still straight intrahepatic arterial branches ("double channel" aspect) have to be differentiated from dilated intrahepatic bile ducts [31].…”
On the basis of our proposed grading, hepatic VMs can be easily classified in subjects with HHT by Doppler US. Depending on the degree of hepatic vascular derangement, appropriate programs for follow up and/or therapy can be designed.
“…To keep labeled subjects with these minimal anomalies make them being periodically checked. Grade 2 VMs with dilated and still straight intrahepatic arterial branches ("double channel" aspect) have to be differentiated from dilated intrahepatic bile ducts [31].…”
On the basis of our proposed grading, hepatic VMs can be easily classified in subjects with HHT by Doppler US. Depending on the degree of hepatic vascular derangement, appropriate programs for follow up and/or therapy can be designed.
“…This was hypothesized to be secondary to hepatic fibrosis and cirrhosis with subsequent increased hepatic arterial blood supply. 15 Although it is probable that all normal gallbladders will not contract rapidly in response to feeding, in those patients in whom rapid contraction of a normal size gallbladder is seen, the diag~ nosis of biliary atresia is extremely unlikely and a liver biopsy rather than radionuclide study may be indicated.…”
Diagnostic ultrasonography plays a major role in the evaluation of jaundiced infants. Seventeen such infants were studied in both fasting and post-prandial states. The gallbladder and biliary tract were evaluated to define features most useful in distinguishing children with biliary atresia from those with other causes of jaundice. Demonstration of a decrease in gallbladder size following feeding virtually eliminates the diagnosis of biliary atresia, even in children with abnormal HIDA scans.
“…The sonographic findings of parallel tubular structures in alcoholic hepatitis have been observed in the past resembling the parallel channel sign seen in obstructive jaundice. [1][2][3] This entity of intrahepatic artery dilation has been termed as PPCS by Sumino and coworkers. 4 The parallel tubular structures in this entity are formed by a dilated hepatic arterial branch and an adjacent branch of the portal vein.…”
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