2016
DOI: 10.1002/ajim.22600
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Source‐specific workplace social support and high‐sensitivity C‐reactive protein levels among Japanese workers: A 1‐year prospective cohort study

Abstract: Supervisor support may have beneficial effects on inflammatory markers in working women. Am. J. Ind. Med. 59:676-684, 2016. © 2016 Wiley Periodicals, Inc.

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Cited by 14 publications
(22 citation statements)
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“…Although psychological distress may partially mediate the association, poor coworker support may be independently associated with the side effect of the COVID-19 vaccine. The possible mechanism underlying the association is unclear, but the finding that the lack of social support generally increases systematic inflammations might clarify this mechanism [4, 12, 28, 29]. Mediators and products that cause inflammation in the circulation can affect body systems to cause systemic side effects [30].…”
Section: Discussionmentioning
confidence: 99%
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“…Although psychological distress may partially mediate the association, poor coworker support may be independently associated with the side effect of the COVID-19 vaccine. The possible mechanism underlying the association is unclear, but the finding that the lack of social support generally increases systematic inflammations might clarify this mechanism [4, 12, 28, 29]. Mediators and products that cause inflammation in the circulation can affect body systems to cause systemic side effects [30].…”
Section: Discussionmentioning
confidence: 99%
“…Mediators and products that cause inflammation in the circulation can affect body systems to cause systemic side effects [30]. For instance, a previous study indicated that low coworker support influenced inflammation biomarkers in a group of employees who worked more than 41h per week [12]. It may be plausible that elevated inflammatory responses associated with lack of coworker support may exaggerate an innate immune response to the COVID-19 vaccine [31, 32].…”
Section: Discussionmentioning
confidence: 99%
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“…Several studies have reported associations between adverse work-related psychosocial factors and increased levels of inflammatory markers. Inflammatory markers, including C reactive protein (CRP),19–24 interleukin 6 (IL-6)24 25 and tumour necrosis factor-α (TNF-α), have been implicated in coordinating atherosclerosis 26. Previous meta-analyses27 28 have identified the associations between psychosocial factors and inflammatory markers; however, the findings from those studies were not conclusive because of methodological heterogeneity (eg, conceptualisation or measurement of work-related psychosocial factors, sample compositions and statistical approaches).…”
Section: Introductionmentioning
confidence: 99%
“…Four studies(82,83,86,87) applied Karasek's job demand-control(-support) JDC(-S) model(10,88): Job strain(82,86,87) and workplace social support(86,87) were not prospectively related to CRP. Meanwhile, when the source of social support was specified, high supervisor support (in contrast to coworker support) was associated with lower CRP among women but not men(83). Job demands were not related to fibrinogen and leukocyte count (87) as well as IL-6(82,86).…”
mentioning
confidence: 99%