Spanish Ketogenic Mediterranean diet: a healthy cardiovascular diet for weight loss

Pérez-Guisado, Joaquín; Muñoz-Serrano, A.; Alonso-Moraga, Ángeles

doi:10.1186/1475-2891-7-30

Cited by 67 publications

(69 citation statements)

References 37 publications

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“…Salas Salvado et al [2008] reported that after consumption of 1 L/week of olive oil for one year in hyperlipidemic subjects, lower incidence of elevated TAG was seen. In another study with Mediterranean diet including olive oil as the principal source of fat for 12 weeks TC and TAG levels were reduced [Perez-Guisado et al, 2008]. Our results are not consistent with observations of Salas Salvado et al [2008] and Perez-Guisado et al [2008], which at a higher amount of olive oil and longer study period resulted in a reduction of TC and TAG.…”

Section: Resultscontrasting

confidence: 99%

Effect of Olive Oil with Low Calorie Diet on Blood Lipids in Hyperlipidemic Patients

Noroozi¹,

Zavoshy²,

Jahanihashemi³

2012

Pol. J. Food Nutr. Sci.

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Key words: olive oil, low calorie diet, hyperlipidemia, serum lipids Serum lipid abnormality is a risk factor for cardiovascular disease. The aim of this research was to study the effect of olive oil with low--calorie diet on blood lipids in hyperlipidemic patients. The study was done on fi fty eight hyperlipidemic patients of both sexes and age range of 25-65 years. The patients received low-calorie diet based on 1400 kcal energy per day for 4 weeks, containing 32% fat, 18% protein and 50% carbohydrate. The intervention group received low-calorie diet including 30 g/d olive oil. Weight, body mass index, waist and hip circumferences were signifi cantly reduced (p<0.05) after 4 weeks in both groups. Treatment with olive oil was associated with a signifi cant reduction (p<0.05) in low density lipoprotein.

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Section: Resultscontrasting

confidence: 99%

Effect of Olive Oil with Low Calorie Diet on Blood Lipids in Hyperlipidemic Patients

Noroozi¹,

Zavoshy²,

Jahanihashemi³

2012

Pol. J. Food Nutr. Sci.

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“…48,49,50,51,52,53 Studies are also now emerging to show the efficacy, sustainability and other positive effects of lowcarbohydrate diets, especially in those with metabolic disease. 54,55,56,57,58,59,60,61 The definition of a low-carbohydrate diet varies in the literature, but most in the field agree that anything over 130 g -150 g per day is not low carbohydrate. 62 An amount of less than 50 g of carbohydrate per day is considered very low carbohydrate, which would put most adults into nutritional ketosis.…”

Section: Trials Of Low-carbohydrate Diets: Significant Findings For Amentioning

confidence: 99%

A low-carbohydrate survey: Evidence for sustainable metabolic syndrome reversal

Cucuzzella

Tondt

Dockter

et al. 2017

JMH

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Background: Metabolic syndrome has become a significant problem, with the American Diabetes Association estimating the cost of diabetes and pre-diabetes in the United States alone to be $322 billion per year. Numerous clinical trials have demonstrated the efficacy of low-carbohydrate diets in reversing metabolic syndrome and its associated disorders.Aim: This study was designed to examine how voluntary adherents to a low-carbohydrate diet rate its effectiveness and sustainability using an online survey.Setting and methods: The 57-question survey was administered online and shared internationally via social media and ‘low-carb’ communities. Where appropriate, chi-squared tests and paired t-tests were used to analyse the responses.Results: There were 1580 respondents. The majority of respondents had consumed less than 100 g of carbohydrates per day for over a year, typically for reasons of weight loss or disease management. There was a reported decrease in waist circumference and weight with a simultaneous decrease in hunger and increase in energy level. Of those who provided laboratory values, the majority saw improvements in their HbA1c, blood glucose measurements, and lipid panel results. There was a reduction in usage of various medications, and 25% reported medication cost savings, with average monthly savings of $288 for those respondents. In particular, the usage of pain relievers and anti-inflammatories dropped with a simultaneous decreased rating of pain and increase in mobility.Conclusion: We conclude that low-carbohydrate diets are a sustainable method of metabolic syndrome reversal in a community setting.

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“…In obese individuals [149] and healthy subjects who underwent eccentric exercise training [150], the inflammatory state was further augmented postprandially through a high CHO intake, but not through high-fat, low CHO meals in the latter study. Maybe more importantly, even moderate CHO restriction has been shown to effectively target several important markers of atherosclerosis and type II diabetes, both of which are associated with chronic inflammation [151][152][153][154][155][156][157]. Forsythe et al showed that in overweight individuals with dyslipidemia a very low CHO diet had a more favorable effect than a low fat diet in reducing several markers of inflammation [158].…”

Section: Avoidance Of Chronic Inflammationmentioning

confidence: 99%

Nutrition Support To Patients Undergoing Gastrointestinal Surgery

Coles¹

2013

Clinical Nutrition

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Over the last years, evidence has accumulated suggesting that by systematically reducing the amount of dietary carbohydrates (CHOs) one could suppress, or at least delay, the emergence of cancer, and that proliferation of already existing tumor cells could be slowed down. This hypothesis is supported by the association between modern chronic diseases like the metabolic syndrome and the risk of developing or dying from cancer. CHOs or glucose, to which more complex carbohydrates are ultimately digested, can have direct and indirect effects on tumor cell proliferation: first, contrary to normal cells, most malignant cells depend on steady glucose availability in the blood for their energy and biomass generating demands and are not able to metabolize significant amounts of fatty acids or ketone bodies due to mitochondrial dysfunction. Second, high insulin and insulin-like growth factor (IGF)-1 levels resulting from chronic ingestion of CHO-rich Western diet meals, can directly promote tumor cell proliferation via the insulin/IGF1 signaling pathway. Third, ketone bodies that are elevated when insulin and blood glucose levels are low, have been found to negatively affect proliferation of different malignant cells in vitro or not to be usable by tumor cells for metabolic demands, and a multitude of mouse models have shown antitumorigenic properties of very low CHO ketogenic diets. In addition, many cancer patients exhibit an altered glucose metabolism characterized by insulin resistance and may profit from an increased protein and fat intake. In this review, we address the possible beneficial effects of low CHO diets on cancer prevention and treatment. Emphasis will be placed on the role of insulin and IGF1 signaling in tumorigenesis as well as altered dietary needs of cancer patients.

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Spanish Ketogenic Mediterranean diet: a healthy cardiovascular diet for weight loss

Cited by 67 publications

References 37 publications

Effect of Olive Oil with Low Calorie Diet on Blood Lipids in Hyperlipidemic Patients

Effect of Olive Oil with Low Calorie Diet on Blood Lipids in Hyperlipidemic Patients

A low-carbohydrate survey: Evidence for sustainable metabolic syndrome reversal

Nutrition Support To Patients Undergoing Gastrointestinal Surgery

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