“…More recently, blood-brain barrier crossover issues, as measured in prairie voles, have been raised by Carter, Boone, Pournajafi-Nazarloo, and Bales (2009), whereby Pitocin exposure may trigger differential exposure to fetal hypoxia via alternations in maternal uterine contraction intensity. Pitocin induction and fetal distress (Akoury, 1991;Bidgood & Steer, 1987;Satin, 1992) has been implicated in fetal neurological impairment (Bors-Koefoed et al, 1998;Fein et al, 1997) and considered in autism (Fein et al, 1997, Waterhouse, Fein, & Modahl, 1996), yet overlooked with respect to ADHD sequelae. Prenatal stress can injure the immature fetal brain, causing significant neurological impairment (Hirst, Walker, Yawno, & Palliser, 2009).…”