Stevens‐Johnson syndrome and abnormal root development: a case report

Brook, U

doi:10.1111/j.1365-263x.1994.tb00112.x

Cited by 10 publications

(6 citation statements)

References 7 publications

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“…Dental growth abnormalities have been reported in patients who developed SJS/TEN during childhood . These include dental agenesis, root dysmorphia, short root, microdontia and incomplete root apex closure .…”

Section: Oral and Dental Sequelaementioning

confidence: 99%

Long-term complications of Stevens-Johnson syndrome/toxic epidermal necrolysis (SJS/TEN): the spectrum of chronic problems in patients who survive an episode of SJS/TEN necessitates multidisciplinary follow-up

2017

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Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) are severe adverse reactions to drugs that cause a life-threatening eruption of mucocutaneous blistering and epithelial sloughing. While the acute complications of SJS/TEN are well described, it is increasingly recognized that survivors may develop delayed sequelae, some of which can be associated with significant morbidity. Studies of long-term SJS/TEN outcomes mostly focus on mucocutaneous and ocular complications. However, other internal organs, such as the respiratory tract and gastrointestinal tract, can be affected. Psychological sequelae are also frequent following the trauma of widespread epidermal necrolysis. An appreciation of the 'chronic' phase of SJS/TEN is needed by clinicians caring for individuals who have survived the acute illness. This review aims to provide an update on the breadth and range of sequelae that can affect patients in the months and years following an acute episode of SJS/TEN.

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Section: Oral and Dental Sequelaementioning

confidence: 99%

Long-term complications of Stevens-Johnson syndrome/toxic epidermal necrolysis (SJS/TEN): the spectrum of chronic problems in patients who survive an episode of SJS/TEN necessitates multidisciplinary follow-up

2017

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“…Intra-oral findings in adults are limited to soft tissue, which can how ulcerative lesions and blisters [8]. However, intra-oral symptoms in growing children are not limited to soft tissue but also pose disadvantages to tooth development [8][9][10][11][12][13]. Currently, only a few foreign studies about developmental disturbance of the permanent teeth related on to SJS have been reported, and no domestic studies have been published so far.…”

Section: ⅰ Introductionmentioning

confidence: 99%

Stevens-Johnson Syndrome : A Case Report

Song¹,

Lee²,

Lee³

et al. 2017

THE JOURNAL OF THE KOREAN ACADEMY OF PEDTATRIC DENTISTRY

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Stevens-Johnson syndrome (SJS), an extremely severe acute hypersensitivity reaction, causes extensive necrosis on the skin and the mucous membrane. SJS is a disease of unknown cause that can occur in all age groups. It is thought to be caused by drug allergy or induced by bacterial infection. Epidermal surface invasion of less than 10 percent is called SJS, and invasion of more than 30 percent is called toxic epidermal necrolysis. Although it is rare with an incidence of 1-2 cases per million people per year, it has effects on tooth development and therefore on children who are in a growth phase. The purpose of this case report is to examine the effect of SJS on tooth development in children. In general, eruption of the upper and lower 1st molars and lower central incisors starts at 6-7 years of age. Root development also occurs at this time. In the case reported here, SJS occurred in a 6-year-old patient. Although the patient's SJS was completely cured , he still suffers from aftereffects. Developmental abnormalities in the patient's teeth were observed only in teeth for which root development had been completed at the time. The purpose of this case report is to illustrate how to diagnose such systemic diseases by intra-oral features and to recognize and resolve tooth development problems associated with the disease.

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“…The acute pathologic changes are well-studied and categorized into various stages, including ulceration with a fibrinous exudative inflammation, ultimately leading to re-epithelialization and healing [56]. The first chronic oral complications were described in the 1980s through several case reports demonstrating permanent loss of tongue papillae with scarring of the oral mucosa, abnormal root development, and hypoplasia of permanent teeth [57][58][59][60][61]. However, it was not until the 2000s that the serious and relatively frequent chronic oral complications of b u r n s x x x ( 2 0 1 5 ) x x x -x x x SJS/TEN were well described.…”

Section: Oralmentioning

confidence: 99%