Sticking together? Re-binding previous other-associated stimuli interferes with self-verification but not partner-verification

Constable, Merryn; Knoblich, Günther

doi:10.1016/j.actpsy.2020.103167

Cited by 7 publications

(5 citation statements)

References 56 publications

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“…This suggests that the referential association of partner is easier to be detached and reassigned to another identity, possibly not originating primarily from the memory enhancement. However, it is important to note that the study design used by Constable and Knoblich 22 differed from our designs in many ways, as described above, and has not always been successful in replicating the partner-advantage (e.g., Constable and colleagues 8 ). Thus it may be inappropriate to directly generalize their findings to the present study.…”

Section: Discussionmentioning

confidence: 92%

“…A similar switch cost was not observed for partner-related shapes. Constable and Knoblich 22 incorporated a joint task and a new “partner” into their experimental design to investigate the memory hypothesis of the identity-prioritization effect. When the identity-shape pair was switched after the participants were familiarized with the pairing, a switch cost to self-related trials was incurred; that is, the response times to the new shape associated with self became significantly longer than response times to the old shape.…”

Section: Discussionmentioning

confidence: 99%

“…Further, the occurrence of partner-advantage is vulnerable to paradigm design, while self-advantage remains dominant across different task settings and stimulus designs. As discussed above, partner-advantage is absent if a joint task includes no-go trials and self is included as a reference stimulus (Experiment 3, Cheng & Tseng, 2019 1 ), or if the shape-matching task contains more identities (five identities in Constable & Knoblich, 2020 22 ). An extensive literature has revealed that the identity processes related to significant others are more malleable than self-advantage.…”

Section: Discussionmentioning

confidence: 99%

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Social affiliation is sufficient to provoke the partner-advantage

Tseng

Jingling

Cheng

2022

Sci Rep

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The partner-advantage is a type of identity-priority processing that we afford to a person with whom we perform a task together 1. The partner-advantage has been revealed by shortened reaction time (RT) and enhanced accuracy when participants learned to match a shape with an associated name. It is distinguished from other long-lasting and robust identity advantages (e.g., self-advantage and friend-advantage) by its instantaneous build-up and quick reduction; however, its characteristics and enabling factors remain unknown. The present study addresses these questions. In Experiment 1, we replicated the partner-advantage in a solo shape-name matching task (i.e., without a social component) in which other identity biases are usually reported. In Experiment 2, an absent partner (who did not appear physically) was sufficient to induce beneficial partner-related processing, with a temporary partner enjoying a benefit similar to that of significant others. In Experiment 3, an identity low in socially affiliated significance (e.g., another participant in the same experiment) did not automatically enjoy a priority bias. Taken together, our results suggest that the bias toward partners, similar to other known identity biases, does not require physical presence to build and maintain a referential advantage. The partner-advantage does not automatically extend to other social affiliations, and a joint task is not a pre-requisite to produce the bias. Our study offers new insights on identity-referential processing and its underlying mechanisms.

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Section: Discussionmentioning

confidence: 92%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Social affiliation is sufficient to provoke the partner-advantage

Tseng

Jingling

Cheng

2022

Sci Rep

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“…3) The trial-level data is either openly available or declared to be obtainable upon request, enabling us to estimate at least one reliability index. Among the 13 papers included, 7 papers made their trial-level data publicly available (Constable & Knoblich, 2020;Constable et al, 2021;Golubickis & Macrae, 2021;Navon & Makovski, 2021;Qian et al, 2020;Schäfer & Frings, 2019;Svensson et al, 2022). For the remaining 6 papers, we reached out to the authors and requested access to their trial-level data.…”

Section: Datasets Acquisitionmentioning

confidence: 99%

A Multiverse Assessment of the Reliability of the Self Matching Task as a Measurement of the Self-Prioritization Effect

Liu,

Hu,

Zheng

et al. 2023

Preprint

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The Self Perceptual Matching Task (SPMT) is a widely used task to investigate the cognitive processes underlying the Self-Prioritization Effect (SPE), wherein performance is enhanced for self-associated stimuli compared to other-associated ones. Despite the wide use of SPMT, there is a lack of attention on its reliability assessment. This ignorance is concerning, given the prevalence of the reliability paradox in cognitive tasks: cognitive tasks demonstrate relatively low reliability when evaluating individual differences, though they produce robust experimen- tal effects. To fill this gap, this preregistered study investigated the reliability of SPMT using a multiverse approach, combining all possible indicators and baselines used to quantify SPE in SPMT. We examined the robustness and the reliability of 24 SPE measures across 17 datasets (N = 805). More specifically, we used a meta-analytical approach to estimate the robustness of SPE across datasets. We calculated the Split-Half Reliability (r) and Intraclass Correla- tion Coefficient (ICC2) for each SPE measure. Our findings revealed a robust experimental effect of SPE across datasets. However, when it came to individ- ual differences, SPE measures derived from Reaction Time (RT) and Efficiency exhibited relatively higher, compared to other SPE measures, but still unsatis- fied split-half reliability (approximately 0.6). Similarly, for the reliability across multiple time points, as assessed by ICC2, RT and Efficiency demonstrated low levels of test-retest reliability (close to 0.5). These findings uncovered the pres- ence of a reliability paradox in the context of SPMT-based SPE assessment. We discussed the implications of our findings for future studies.

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“…The nucleus pulposus secretes pro-inflammatory molecules, such as tumor necrosis factor (TnF)-α, interleukin (il)-1β, IL-6 and IL-17, of which TnF-α is the most prominent, promoting the degradation of ecM, and leading to cell phenotypic changes and a series of degenerative events (8). in addition, endoplasmic reticulum (er) stress is also one of the potential factors for the induction of idd by inducing nPc apoptosis and ecM degradation (9). Thus, finding effective drugs to inhibit ecM degradation, inflammatory response and ER stress-induced apoptosis in HnPcs may be a feasible strategy for the prevention and treatment of idd.…”

Section: Introductionmentioning

confidence: 99%

Hyperoside ameliorates TNF‑α‑induced inflammation, ECM degradation and ER stress‑mediated apoptosis via the SIRT1/NF‑κB and Nrf2/ARE signaling pathwaysin vitro

et al. 2022

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intervertebral disc degeneration (idd) is the main pathogenesis of numerous cases of chronic neck and back pain, and has become the leading cause of spinal-related disability worldwide. Hyperoside is an active flavonoid glycoside that exhibits anti-inflammation, anti-oxidation and anti-apoptosis effects. The purpose of the present study was to investigate the effect of hyperoside on tumor necrosis factor (TnF)-α-induced idd progression in human nucleus pulposus cells (nPcs) and its potential mechanism. The activity and apoptosis of nPcs were detected by cell counting Kit-8 and flow cytometry analyses, respectively. The expression of interleukin (il)-6 and il-1β was detected with eliSa kits. Western blotting was used to detect the expression levels of proteins. The results showed that hyperoside effectively alleviated TnF-α-induced nPc apoptosis, and hyperoside treatment inhibited the upregulation of inducible nitric oxide synthase, cyclooxygenase-2, il-1β and il-6 in TnF-α-stimulated nPcs. Compared with the findings in the TNF-α group, the intervention of hyperoside attenuated the upregulated expression of aggrecan and collagen ii, and downregulated the expressions of matrix metalloproteinase (MMP) 3, MMP13 and a disintegrin and metalloproteinase with thrombospondin motifs 5. in addition, hyperoside upregulated sirtuin-1 (SirT1) and nuclear factor e2-related factor 2 (nrf2) protein expression, and inhibition of SirT1 or nrf2 signaling reversed the protective effect of hyperoside on TnF-α-induced nPcs. in summary, hyperoside ameliorated TnF-α-induced inflammation, extracellular matrix degradation, and endoplasmic reticulum stress-mediated apoptosis, which may be associated with the regulation of the SirT1/nF-κB and nrf2/antioxidant responsive element signaling pathways by hyperoside.

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Sticking together? Re-binding previous other-associated stimuli interferes with self-verification but not partner-verification

Cited by 7 publications

References 56 publications

Social affiliation is sufficient to provoke the partner-advantage

Social affiliation is sufficient to provoke the partner-advantage

A Multiverse Assessment of the Reliability of the Self Matching Task as a Measurement of the Self-Prioritization Effect

Hyperoside ameliorates TNF‑α‑induced inflammation, ECM degradation and ER stress‑mediated apoptosis via the SIRT1/NF‑κB and Nrf2/ARE signaling pathwaysin vitro

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