Stomatococcus mucilaginosus bacteremias: Typical case presentations, simplified diagnostic criteria, and a literature review

Localization of the Genus Rothia in the Oral Cavity

“…Furthermore, this organism is a responsible pathogen for a variety of opportunistic infections (11,(15)(16)(17)(18). Immunocompromised patients are very often infected with this pathogen, leading to endocarditis (11,12,17,19), sepsis (20,21), peritonitis (17), and infections associated with tissue and organ transplantations and implants. Complications are also caused in patients with tumors, diabetes, and other diseases (22).…”

<i>Rothiamucilaginosa</i> life threatening infections in non-neutropenic hosts

“…Infections due to this commonly believed low virulence organism, have been rarely reported in non-neutropenic patients and always has been associated with pre-existing risk factors. These include profound immune-compromise states, prophylactic antibiotic use, repeated broad-spectrum antibiotics exposure, mucositis, injectable drug use, abnormal cardiac valves, and chronic indwelling catheter infections [4][5][6][7][8][9][10][11]. Taxonomic confusion and misidentification with the common skin contaminants like staphylococcus, streptococcus or micrococcus, can lead to serious complications and delays in treatment.…”

A selective medium for Rothia mucilaginosa and its distribution in oral cavities

“…Furthermore, the organism is responsible for a variety of opportunistic infections (Pinsky et al, 1989;Lemozy et al, 1990;Magee et al, 1990;Mitchell et al, 1990;Abraham et al, 1997). Immunocompromised patients are often infected with this pathogen, leading to endocarditis (Pinsky et al, 1989;Rubin et al, 1978;Mitchell et al, 1990;Coudron et al, 1987), sepsis (Barlow et al, 1986;Poirier and Gaudereau, 1989), and peritonitis (Mitchell et al, 1990). Complications have also been reported in patients with tumors, diabetes, and other diseases (Patey et al, 1991).…”

“…R. mucilaginosa were isolated in several laboratories, mostly from the oral cavity, throat, dental plaques, and persistent apical periodontitis (Gordon, 1967;Pinsky et al, 1989;Rubin et al, 1978;Bowden, 1969;Yamane et al, 2010). Furthermore, the organism is responsible for a variety of opportunistic infections (Pinsky et al, 1989;Lemozy et al, 1990;Magee et al, 1990;Mitchell et al, 1990;Abraham et al, 1997). Immunocompromised patients are often infected with this pathogen, leading to endocarditis (Pinsky et al, 1989;Rubin et al, 1978;Mitchell et al, 1990;Coudron et al, 1987), sepsis (Barlow et al, 1986;Poirier and Gaudereau, 1989), and peritonitis (Mitchell et al, 1990).…”