Studies on Snakebites in the Asian Areas

Sawai, Yoshio

doi:10.1016/b978-0-08-024952-0.50007-7

Cited by 11 publications

(12 citation statements)

References 9 publications

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“…Severe local edema is a relevant clinical finding in Bothrops snake-bitten victims (55, 56) that frequently leads to ischemia and neural compression, contributing to tissue loss and disability (57–59). Experimental studies on the dynamics of local inflammation caused by B. asper venom have demonstrated early plasma extravasation from small venules and adjoining capillary segments a few minutes after exposure to venom, followed by stasis in the microcirculation (60).…”

Section: Inflammation Induced By Viper Snake Venommentioning

confidence: 99%

“…A conspicuous infiltration of leukocytes into the local snakebite is another important characteristic of inflammation triggered by the majority of Viperidae viperid snake venoms (55, 56). Leukocytes are essential cells in host defense, and they respond to injury by releasing inflammatory mediators, performing phagocytosis and inducing production of potent microbicidal products.…”

Section: Inflammation Induced By Viper Snake Venommentioning

confidence: 99%

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Inflammation Induced by Platelet-Activating Viperid Snake Venoms: Perspectives on Thromboinflammation

et al. 2019

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Envenomation by viperid snakes is characterized by systemic thrombotic syndrome and prominent local inflammation. To date, the mechanisms underlying inflammation and blood coagulation induced by Viperidae venoms have been viewed as distinct processes. However, studies on the mechanisms involved in these processes have revealed several factors and signaling molecules that simultaneously act in both the innate immune and hemostatic systems, suggesting an overlap between both systems during viper envenomation. Moreover, distinct classes of venom toxins involved in these effects have also been identified. However, the interplay between inflammation and hemostatic alterations, referred as to thromboinflammation, has never been addressed in the investigation of viper envenomation. Considering that platelets are important targets of viper snake venoms and are critical for the process of thromboinflammation, in this review, we summarize the inflammatory effects and mechanisms induced by viper snake venoms, particularly from the Bothrops genus, which strongly activate platelet functions and highlight selected venom components (metalloproteases and C-type lectins) that both stimulate platelet functions and exhibit pro-inflammatory activities, thus providing insights into the possible role(s) of thromboinflammation in viper envenomation.

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Section: Inflammation Induced By Viper Snake Venommentioning

confidence: 99%

Section: Inflammation Induced By Viper Snake Venommentioning

confidence: 99%

Inflammation Induced by Platelet-Activating Viperid Snake Venoms: Perspectives on Thromboinflammation

et al. 2019

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“…However, only w15% of snakebite cases are treated with antivenom, with the majority being treated with herbal medicine. 1 In the 1980s, horse immunoglobulin G-derived antivenom made with several types of F(ab 0 ) 2 fragments were developed by the Vaccine Center, Center for Disease Control, Taiwan. 2e4 These included a bivalent antivenom against the hemorrhagic venom (FH antivenom) of the green habu (Viridovipera stejnegeri) and the Taiwan habu (Protobothrops mucrosquamatus); a bivalent antivenom against the neurotoxic venom (FN antivenom) of the Taiwan banded krait (Bungarus multicinctus) and the Chinese cobra (Naja atra); and an antivenom against the hemorrhagic venom of the hundredpace snake (FA antivenom; Deinagkistrodon acutus).…”

Section: Introductionmentioning

confidence: 99%

An investigation of snakebite antivenom usage in Taiwan

Lin

Chaou

Tseng

2016

Journal of the Formosan Medical Association

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“…The present study unconsciously takes a different stand from the dominating or conventional research going on in the discipline. Figure 1 may help us understand the design of this commentary review kind of study, especially Bhat, 1974;Gaitonde & Bhattacharya, 1980;Hati et al,1992;Kulkarni & Annees, 1994;Saha & Hati, 1983;Sawai & Honma, 1975 Lim, 1980;Reid, 1963;Sawai et al, 1978;Singh, 1980 Thailand 0.9 1.5-6.6 Buranasin, 1993;Sawai,1973Sawai, ,1980Trishnananda, 1979 0.01-0.14 WHO, 2005 Source: The authors.…”

Section: Conceptual Framework and Propositionmentioning

confidence: 99%

Fang Under ‘Red Tape’: A Barrier for Underutilisation of Human Resource (Case of Snake Charmers, India)

Halder¹,

Paul

2022

South Asian Survey

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The vulnerability of human death due to snakebite could (possibly) be countered in the most feasible way of utilising snakes themselves if properly organised. This article attempts to identify an indigenous community (snake charmer) with its inherent expertise for answering a large-scale health issue of snakebite. Existing bureaucratic directives often do barricade such human resources from their proper utilisation through stringent laws and regulations. In the case of a tropical developing country like India, snake charmers may be the capital of captivating the ladder of betterment when governments treat them as assets. Examining snakebite fatality over the globe and gradually zooming into a state level (West Bengal) prone to health hazards, the article sounds true to its venture of showcasing a policy orientation for minimising human deaths and disadvantages. In place of a barrier, ’red tape’ might be acting as a bridge of development between wildlife and humanity.

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Studies on Snakebites in the Asian Areas

Cited by 11 publications

References 9 publications

Inflammation Induced by Platelet-Activating Viperid Snake Venoms: Perspectives on Thromboinflammation

Inflammation Induced by Platelet-Activating Viperid Snake Venoms: Perspectives on Thromboinflammation

An investigation of snakebite antivenom usage in Taiwan

Fang Under ‘Red Tape’: A Barrier for Underutilisation of Human Resource (Case of Snake Charmers, India)

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