“…4) This complication could occur in the left main coronary artery, in the RCA, or in both. Several pathophysiologic mechanisms have been suggested, and these include coronary spasm, debris/ thrombus embolization from the aortic valve or wall, [5][6][7] and intraoperative iatrogenic causes, such as extraluminal compression (e.g., the surgical material, the suture used to close, hematoma) 8) or vessel transection (e.g., vessel kinking). 4,9) The usual symptoms and clinical findings of COS after SAVR include the development of unstable angina, which could be treated by either CABG 1,2) or PCI.…”