“…As a consequence, the precursor pepsinogen cannot be converted into its active form pepsin, and therefore peptic digestion is inhibited. Additionally, sucralphate has pepsin‐binding capacity, which supports the effect of hindering digestion [7, 8]. Furthermore, sucralphate contains aluminium, which independently leads to an increased probability of sensitization when applied parenterally [9] or via the oral route [10].…”