2017
DOI: 10.1177/1751143717732729
|View full text |Cite
|
Sign up to set email alerts
|

Sudden cardiac arrest in hypertrophic cardiomyopathy with dynamic cavity obstruction: The case for a decatecholaminisation strategy

Abstract: Catecholamines are entrenched in the management of shock states. A paradigm shift has pervaded the critical care arena in recent years acknowledging their propensity to cause harm and fuel a 'death-spiral'. We present the case of a 21-yearold male following a witnessed out-of-hospital cardiac arrest who received high-quality cardiopulmonary resuscitation and standard advanced life support for refractory ventricular fibrillation until return of spontaneous circulation after 70 min. Early post-admission echocard… Show more

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1

Citation Types

0
1
0

Year Published

2019
2019
2019
2019

Publication Types

Select...
2

Relationship

0
2

Authors

Journals

citations
Cited by 2 publications
(1 citation statement)
references
References 53 publications
(69 reference statements)
0
1
0
Order By: Relevance
“…An awareness that epinephrine may sometimes be proarrhythmic in other acute cardiac deterioration is important; examples may include myocarditis or acute myocardial ischemia (such as due to anomalous coronary arterial supply). Epinephrine may also induce paradoxical hypotension during the acute management of cardiac arrest due to obstructive hypertrophic cardiomyopathy (28). In such cases, β1-adrenergic effects increase contractility and thus the outflow obstruction, whereas β2-adrenergic effects reduce systemic vascular resistance.…”
Section: Discussionmentioning
confidence: 99%
“…An awareness that epinephrine may sometimes be proarrhythmic in other acute cardiac deterioration is important; examples may include myocarditis or acute myocardial ischemia (such as due to anomalous coronary arterial supply). Epinephrine may also induce paradoxical hypotension during the acute management of cardiac arrest due to obstructive hypertrophic cardiomyopathy (28). In such cases, β1-adrenergic effects increase contractility and thus the outflow obstruction, whereas β2-adrenergic effects reduce systemic vascular resistance.…”
Section: Discussionmentioning
confidence: 99%