Sudden hearing loss with simultaneous posterior semicircular canal BPPV: Possible etiology and clinical implications

El‐Saied, Sabri; Joshua, Ben‐Zion; Segal, N; Kraus, Mordechai; Kaplan, Daniel M.

doi:10.1016/j.amjoto.2013.08.021

Cited by 23 publications

(15 citation statements)

References 20 publications

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“…Based on these cases, the authors proposed viral labyrinthitis rather than a vascular infarct. Conversely, El-Saied et al 46 described five patients with hearing loss and BPPV and proposed a vascular insult as the common pathophysiological mechanism. 46 It seems that when BPPV is associated with sudden hearing loss, treatment requires several sessions of a repositioning maneuver.…”

Section: Idiopathic Sudden Sensorineural Hearing Lossmentioning

confidence: 99%

Review of the pathology underlying benign paroxysmal positional vertigo

Yetışer

2019

J Int Med Res

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The pathophysiological mechanism underlying benign paroxysmal positional vertigo (BPPV) is related to free-floating debris/otoliths in the semicircular canal (canalolithiasis) or debris/otoliths attached to the cupula (cupulolithiasis). These debris/otoliths are considered to originally accumulate after detachment from the neuroepithelium of the utricular macula secondary to a type of degeneration. An idiopathic form, which is assumed to occur spontaneously, is diagnosed when the causative pathology is obscure. However, an association between various other systemic or inner ear conditions and BPPV has been reported, indicating the existence of secondary BPPV. This study was performed to present the first review of the pathology underlying BPPV following a complete PubMed/Medline search. In total, 1932 articles published from 1975 to 2018 were reviewed. The articles were classified according to 17 potentially causative factors (aging; migraine; Meniere's disease; infection; trauma; idiopathic sudden sensorineural hearing loss; sleeping habits; osteoporosis and vitamin D insufficiency; hyperglycemia and diabetes mellitus; chronic head and neck pain; vestibule or semicircular canal pathology; pigmentation disorders; estrogen deficiency; neurological disorders; autoimmune, inflammatory, or rheumatologic disorders; familial or genetic predisposition; and allergy). A discussion of the underlying cause of BPPV for each factor is presented.

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Section: Idiopathic Sudden Sensorineural Hearing Lossmentioning

confidence: 99%

Review of the pathology underlying benign paroxysmal positional vertigo

Yetışer

2019

J Int Med Res

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“…Pathophysiologic mechanism of positional nystagmus in SSNHL Previous reports on SSNHL with concomitant BPPV suggested that selective damage of the cochlea, which results in SSNHL, and the utricle or saccule, which causes otoconial release, may lead to development of this condition (Baloh et al, 1987;Rambold et al, 2004;El-Saied et al, 2014). Viral inflammation within the inner ear may cause this 'patchy' pattern of inner ear dysfunction, which was suggested as the most likely explanation for the selective damage of the cochlea and otolithic organs without damaging the SCCs .…”

Section: Classification Of Positional Nystagmusmentioning

confidence: 95%

“…SSNHL often accompanies vertigo, and BPPV has been simultaneously observed in 8-19% of all SSNHL patients Kim & Ban, 2012;Kim et al, , 2014aSong et al, 2012;Hong & Yeo, 2013;El-Saied et al, 2014). BPPV was reported to most commonly involve the posterior semicircular canal (PSCC; 53%), followed by the lateral semicircular canal (LSCC; 25%), both PSCC and LSCC (19%), and the anterior semicircular canal (ASCC; 3%) (Song et al, 2012).…”

Section: Classification Of Positional Nystagmusmentioning

confidence: 98%

Sudden sensorineural hearing loss with positional vertigo: Initial findings of positional nystagmus and hearing outcomes

Kim

Shin

Yang

et al. 2016

International Journal of Audiology

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“…Furthermore, the disease can develop via inner ear disorders which ultimately lead to the degradation and disassociation of otoconia from their native gelatinous substrate. These include vestibular neuritis, Meniere's disease and sudden sensorineural hearing loss (SNHL) …”

Section: Pathophysiologymentioning

confidence: 99%

“…These include vestibular neuritis, Meniere's disease and sudden sensorineural hearing loss (SNHL). [15][16][17] BPPV can develop in any of the three semicircular canals. The posterior canal variant is by far the most common (80-90%) 18 because it is the most gravitydependent part of the vestibular labyrinth.…”

Section: Pathophysiologymentioning

confidence: 99%

Benign paroxysmal positional vertigo

You

Instrum

Parnes

2018

Laryngoscope Investig Oto

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Objectives Benign paroxysmal positional vertigo (BPPV) is the most common peripheral vestibular end‐organ disease. This article aims to summarize research findings and key discoveries of BPPV. The pathophysiology, diagnosis, nonsurgical, and surgical management are discussed. Methods A comprehensive review of the literature regarding BPPV up through June 2018 was performed. Results BPPV is typified by sudden, brief episodes of vertigo precipitated by specific head movements. While often self‐limited, BPPV can have a considerable impact on quality of life. The diagnosis can be established with a Dix‐Hallpike maneuver for the posterior and anterior canals, or supine roll test for the horizontal canal, and typically does not require additional ancillary testing. Understanding the pathophysiology of both canalithiasis and cupulolithiasis has allowed for the development of various repositioning techniques. Of these, the particle repositioning maneuver is an effective way to treat posterior canal BPPV, the most common variant. Options for operative intervention are available for intractable cases or patients with severe and frequent recurrences. Conclusions A diagnosis of BPPV can be made through clinical history along with diagnostic maneuvers. BPPV is generally amenable to in‐office repositioning techniques. For a small subset of patients with intractable BPPV, canal occlusion can be considered. Level of Evidence N/A

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Sudden hearing loss with simultaneous posterior semicircular canal BPPV: Possible etiology and clinical implications

Cited by 23 publications

References 20 publications

Review of the pathology underlying benign paroxysmal positional vertigo

Review of the pathology underlying benign paroxysmal positional vertigo

Sudden sensorineural hearing loss with positional vertigo: Initial findings of positional nystagmus and hearing outcomes

Benign paroxysmal positional vertigo

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