Supracristal Ventricular Septal Defect Complicated by Formation of an
                        Aorto-Right Ventricular Outflow Tract Fistula: A Rare Cause of Biventricular
                        Enlargement

Muhyieddeen, Amer; Sadhale, Ashwini; Kunchakarra, Siri; Rathod, Ankit

doi:10.14797/pefd1523

Cited by 2 publications

(2 citation statements)

References 8 publications

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“…The formation of true perimembranous aneurysm is due to the failure to close the fetal membranous ventricular septum in time after birth, the closure of the membranous portion is delayed after birth, and part of the generated membranous portion is continuously pressed under the left ventricular high pressure to form perimembranous aneurysm, leaving the top of the tumor, and the breach causes a left-to-right shunt at the ventricular level. Another common perimembranous aneurysm VSD is the so-called pseudoperimembranous aneurysm, which is a relatively immature fibrous tissue formed by the continuous proliferation and adhesion of the tricuspid valve septum, chordae tendineae, or surrounding tissue [7][8][9]. Previous study has shown that under the condition of left-to-right shunting of intraventricular blood at the level of VSD chamber, long-term high pressure impacted the inferior membrane defect, causing damage to the perimembranous tissue and the gap of the true perimembranous aneurysm.…”

Section: Discussionmentioning

confidence: 99%

[Retracted] Study on Inflammatory Factors in Aneurysmal Perimembranous Ventricular Septal Defect in Congenital Heart Disease

Zhou

Liu

Wang

et al. 2022

BioMed Research International

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To detect the expression of inflammatory factors such as interleukin-1β (IL-1β), interleukin-6 (IL-6), transforming growth factor (TGF-β), and tumor necrosis factor (TNF-α) in the tumor tissue of ventricular septal defect (VSD) in congenital heart disease and to explore the role of inflammatory response in the formation of aneurysmal perimembranous VSD(APVSD). Children with APVSD of congenital heart disease treated by surgery were selected and divided into true aneurysmal perimembranous group (TAP group) and pseudoaneurysmal perimembranous group (PAP group) according to echocardiography and surgical findings. There were 15 children in the TAP group and 31 in the PAP group. The aneurysmal perimembranous tissue of the two groups of children was collected during the operation. IL-1β, IL-6, TGF-β, and TNF-α were positively expressed in the aneurysmal perimembranous tissue of the two groups, and the expression levels of all inflammatory factors in the PAP group were higher than those in the TAP group, and the difference was statistically significant ( P < 0.05 ). The expression levels of IL-1β, IL-6, TGF-β, and TNF-α in the aneurysmal perimembranous tissue of the two groups were negatively correlated with the width of the APVSD breach. IL-1β, IL-6, TGF-β, and TNF-α may be involved in the occurrence and development of APVSD through inflammatory mechanism.

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Section: Discussionmentioning

confidence: 99%

[Retracted] Study on Inflammatory Factors in Aneurysmal Perimembranous Ventricular Septal Defect in Congenital Heart Disease

Zhou

Liu

Wang

et al. 2022

BioMed Research International

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“…19,20 Underassessment and mischaracterization of VSDs, specifically outlet VSDs and peripheral aortic valve anatomy, can lead to missed pathology that would require surgical VSD closure or device closure intervention. 21,22 In addition to aortic valve prolapse and AR, a sinus of Valsalva aneurysm (SOVA) is another condition that can be caused by an outlet VSD that compromises the integrity of the ventricular septal wall. 23,24…”

Section: Discussionmentioning

confidence: 99%

Outlet Ventricular Septal Defect: A Case Report on the Importance of Multi-Window Sonographic Assessment

Kallstrom

2024

Journal of Diagnostic Medical Sonography

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Congenital heart disease (CHD) is the existence of structural heart problems since the formation of the heart prebirth. There are different categories of CHD, all having their own unique frequency and severity characteristics. One type of CHD is a ventricular septal defect (VSD), more specifically outlet VSD. A VSD is the most common congenital heart defect; however, their presentation can be quite complex. Color and spectral Doppler transthoracic echocardiography (TTE) are valuable tools used in the diagnosis of VSDs. The often-challenging presentation of these defects requires sonographers to confirm their existence through the parasternal long-axis, parasternal short-axis, apical four chamber, apical three chamber, and apical five chamber scanning windows. Although VSDs are the most common congenital heart defect, their wide-ranging uniqueness and location along the interventricular septum (IVS) to assess all areas along the IVS on all exams, specifically those that demonstrate left-to-right shunting. The objective of this case report was to stress the importance of multi-window assessment during the sonographer’s interrogation of outlet VSDs, due to the potential implications on adjacent anatomy and the potential for surgical intervention.

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Supracristal Ventricular Septal Defect Complicated by Formation of an Aorto-Right Ventricular Outflow Tract Fistula: A Rare Cause of Biventricular Enlargement

Cited by 2 publications

References 8 publications

[Retracted] Study on Inflammatory Factors in Aneurysmal Perimembranous Ventricular Septal Defect in Congenital Heart Disease

[Retracted] Study on Inflammatory Factors in Aneurysmal Perimembranous Ventricular Septal Defect in Congenital Heart Disease

Outlet Ventricular Septal Defect: A Case Report on the Importance of Multi-Window Sonographic Assessment

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