Surgical Management of Corrosive Gastric Injuries

Nicosia, Jon F.; Thornton, Joseph P.; Folk, Frank A.; Saletta, John D.

doi:10.1097/00000658-197408000-00001

Cited by 42 publications

(15 citation statements)

References 12 publications

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“…There have been reports of malignancy developing in a scarred esophagus or stomach following corrosive ingestion [24–26]. However, in our experience this association has been found to be tenuous.…”

Section: Discussioncontrasting

confidence: 47%

Acute Corrosive Injuries of the Stomach: A Single Unit Experience of Thirty Years

Ananthakrishnan

Parthasarathy

Kate

2011

ISRN Gastroenterology

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Introduction. The spectrum of gastric injury due to corrosives can vary. This paper presents a single center experience of over 30 years of corrosive gastric injuries of 39 patients with acute gastric injuries from 1977 till 2006. Patients and Methods. Two thirds of the patients in the acute injury group had a concomitant esophageal injury. The age of the patients ranged from 4 years to 65 years with a slight preponderance of males. (M : F ratio 22 : 17). Results. 36 out of 39 acute gastric injuries were due to ingestion of acids. Three patients had history of caustic soda ingestion. Oral hyperemia or ulcers of varying extent were seen in all patients. The stomach showed hyperemia in 10, extensive ulcers in 13, and mucosal necrosis in 10 patients. Fifteen patients (15/39, 38.5%) were managed conservatively. Twenty four patients (24/39, 61.5%) underwent laparotomy: one for frank peritonitis, 10 for gastric mucosal necrosis, and 13 others for extensive gastric ulcerations. Overall the mortality rate was 29.6 %. Conclusion. Although the mortality and morbidity of acute corrosive gastric injuries is high, the key to improve the survival is early identification of perforation, maintenance of nutrition and control of sepsis.

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Section: Discussioncontrasting

confidence: 47%

Acute Corrosive Injuries of the Stomach: A Single Unit Experience of Thirty Years

Ananthakrishnan

Parthasarathy

Kate

2011

ISRN Gastroenterology

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“…3 Acid induces coagulative necrosis with eschar formation, and this may limit tissue penetration, but this does not appear to be a major variable in determining the extent of injury. 12,13 Thrombosis of small vessels and production of heat exacerbate the initial corrosive injury. Tissue injury progresses rapidly in the first few minutes but can continue for several hours.…”

Section: Discussionmentioning

confidence: 99%

Ingestion of Caustic Substances by Adults

Satar

Topal

Kozacı

2004

American Journal of Therapeutics

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Caustic products are responsible for the most serious cases of poisoning, which are always emergency cases. In this paper, we review demographic features and endoscopic results of the patients admitted to a university emergency department with a history of caustic substance ingestion between January 2000 and June 2003. Thirty-seven patients were included in this study. Twenty-one of the patients were female and 16 were male. The mean age of the patients was 30.9 +/- 14.7 years. The agents included sodium hypochlorite in 24 patients and hydrochloric acid in 13 patients. All the patients ingested these agents orally. The mean interval time of admission to emergency department after ingestion of caustic agent was 5.4 +/- 5.6 hours. Endoscopy was attempted in 37 patients. Endoscopic results were as follows: grade 0 in 8 (21.6%) patients, grade 1 in 17 (45.9%) patients, grade 2a in 5 (13.5%) patients, and grade 2b in 7 (18.9%) patients. We believe that early signs and symptoms after caustic substance ingestion are not consistent with the extent of damage, and endoscopy is the only reliable method to assess injury. It is important that efforts should be made to educate the public about the dangers of caustic substances so that their threat may be diminished.

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“…It has been generally believed that acids spare the esophagus because of its alkaline environment and the relative resistance of its mucosa to acids and instead result in gastric injuries (8,14). Similarly, gastric injury is believed to be less common with alkalies, which may be partly neutralized by the acidic contents of the stomach (1).…”

Section: Discussionmentioning

confidence: 99%

Radiological spectrum of late sequelae of corrosive injury to upper gastrointestinal tract. a pictorial review

et al. 2004

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Surgical Management of Corrosive Gastric Injuries

Abstract: Four cases of extensive corrosive gastric injury requiring surgical treatment are presented. Appropriate management of this life-threatening entity requires an awareness of its subtle presentation plus a well planned surgical approach.

Cited by 42 publications

References 12 publications

Acute Corrosive Injuries of the Stomach: A Single Unit Experience of Thirty Years

Acute Corrosive Injuries of the Stomach: A Single Unit Experience of Thirty Years

Ingestion of Caustic Substances by Adults

Radiological spectrum of late sequelae of corrosive injury to upper gastrointestinal tract. a pictorial review

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