Surgical Repair of a Perforated Gastric Ulcer in a Foal

Probst, Curtis W.; Schneider, Robert K.; Hubbell, J. A. E.; Hart, Emma C J

doi:10.1111/j.1532-950x.1983.tb00714.x

Cited by 15 publications

(5 citation statements)

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“…There was no seromuscular tear extending beyond the limits of the mucosal tear in these cases, suggesting that the entire thickness of the wall was weakened before the tear. Gastric perforation in foals can be repaired surgically (Probst, Schneider, Hubbell and Hart 1983) if done promptly.…”

Section: )mentioning

confidence: 99%

Gastric rupture in horses: A review of 54 cases

Todhunter

Erb

Roth

1986

Equine Veterinary Journal

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Summary The historical, clinical, laboratory, surgical and necropsy findings in 54 cases of gastric rupture in horses are described. Eleven per cent of the deaths of horses undergoing exploratory coeliotomy for colic during the period of the study were a result of gastric rupture. Comparison with all horses which had exploratory coeliotomies for colic over an eight year period did not show that horses with gastric rupture were different from these reference horses regarding age, breed or season. There were fewer stallions than expected in the gastric rupture group. Horses with histories of both acute and chronic (more than 36 h) colic were susceptible to gastric rupture. Primary and idiopathic causes of gastric dilation and rupture accounted for about one‐third of the horses. All but one of these cases resulting from secondary causes fell into three aetiologically‐related groups: obstructive, peritoneal and enteric, with approximately equal numbers of horses in each group. Most of the ruptures occurred along the greater curvature of the stomach. At least six horses ruptured their stomachs post‐operatively in the presence of an indwelling nasogastric tube. The presence or absence of gastric reflux following nasogastric intubation was not a reliable indicator, on its own, of gastric dilation. Horses that later died from gastric rupture had markedly elevated heart rate, hypochloraemia, peritoneal exudative effusion (particularly with evidence of sepsis), pre‐and/or postoperative gastric reflux and small or large intestinal disease. However, no distinctive feature of these horses was shown to place them at risk of gastric rupture.

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Section: )mentioning

confidence: 99%

Gastric rupture in horses: A review of 54 cases

Todhunter

Erb

Roth

1986

Equine Veterinary Journal

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“…) and the third was perforating gastric ulcer in a foal (Probst et al . ). Prerequisites for successful treatment appear to be early recognition of the rupture before it has progressed and caused peritoneal contamination and the location of the tear must be accessible surgically (Hackett ).…”

Section: Clinical Signsmentioning

confidence: 97%

Review of gastric rupture in the horse

Dechant

Winfield²

2015

Equine Veterinary Education

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Summary Gastrointestinal rupture is an important cause and complication of equine colic. The stomach is the most commonly affected segment in the gastrointestinal tract involved in gastrointestinal rupture. Gastric rupture can be primary or secondary; however, unless prodromal clinical signs are identified, localised and corrected, the resulting peritoneal contamination with feed, intestinal secretions and bacteria is invariably fatal. Causes for gastric rupture may be known or idiopathic; however, factors that predispose a horse to gastric rupture are poorly understood. Further research is needed to identify underlying causes and pathophysiology of gastric rupture to prevent it from occurring.

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“…GASTRIC and/or duodenal ulceration (GDU) is becoming recognised as a clinically significant disease in the foal. While reports of necropsy (Rooney 1964;Rebhun, Dill and Power 1982;Acland, Gunson and Gillette 1983) and surgical findings (Valdez 1979;Wagner, Grant, Schmidt and Hallowell 1979;McIntosh and Shupe 1981 ;Gross and Mayhew 1983;Probst, Schneider, Hubbell and Hart 1983) have helped characterise the pathology involved, little objective data exist about the early diagnosis and successful management of the condition. This review presents an overview of GDU in foals with reference to and extrapolations from research pertaining to human peptic ulcer disease.…”

Section: Introductionmentioning

confidence: 99%

Gastroduodenal ulceration in foals

Becht

Byars²

1986

Equine Veterinary Journal

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Summary Gastroduodenal ulceration is becoming recognised as an important disease ***in ***foals during the first few months of life. Aetiopathogenesis is presumed to be similar to peptic disease in humans associated with back diffusion of hydrogen ions into the mucosa. Many factors have been incriminated as predisposing foals to ulceration but few have been proven. To date, use of non‐steroidal anti‐inflammatory agents has been the only documented cause of gastroduodenal ulceration in foals. The clustering of affected foals on certain farms suggests an infectious aetiology but attempts to identify a causative organism have been unsuccessful. Four clinical syndromes defined for foals with gastroduodenal ulceration include: (1) silent ulcers, which occur most often in the non‐glandular stomach along the margo plicatus and are identified as incidental findings at necropsy; (2) active ulcers which are often manifested by abdominal pain, excessive salivation and bruxism; (3) perforating ulcers which usually result in a severe, diffuse peritonitis; and (4) pyloric or duodenal obstruction from a healing ulcer. General approaches to therapy of a foal with active ulceration consist of reduction of gastric acidity and enhancement of mucosal protection. Antacids and type 2 histamine receptor antagonists are used most often to neutralise or decrease acid secretion, respectively. Sucralfate, a locally active sulphated sucrose preparation, is commonly used as a cytoprotective agent. The efficacy and safety of many products used have not been evaluated adequately in foals. Perforating ulcers are usually associated with death or humane destruction of the foal because of fulminating peritonitis. Surgical intervention and bypass procedures are indicated in foals that develop pyloric or duodenal obstructions from healing ulcers.

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Surgical Repair of a Perforated Gastric Ulcer in a Foal

Cited by 15 publications

References 5 publications

Gastric rupture in horses: A review of 54 cases

Gastric rupture in horses: A review of 54 cases

Review of gastric rupture in the horse

Gastroduodenal ulceration in foals

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