Surgical Treatment of Benign Intracranial Hypertension—Subtemporal Decompression Revisited

Kessler, Laibe A.; Novelli, Paula M.; Reigel, Donald H.

doi:10.1016/s0090-3019(97)00359-5

Cited by 33 publications

(18 citation statements)

References 8 publications

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“…All 14 patients with post-operative follow-up examinations had resolution of papilledema, there were no deaths and no one required a second operation for PTC. A recent series of eight pseudotumor patients treated by subtemporal decompression published in 1998 reconfirmed Dandy's excellent clinical outcomes [3]. Within one month of treatment, deterioration in visual fields and acuity resolved in all eight patients.…”

Section: Subtemporal Decompressionmentioning

confidence: 77%

“…Within one month of treatment, deterioration in visual fields and acuity resolved in all eight patients. Five of eight patients required CSF diversion to control headaches, but no patient experienced recurrent permanent visual deterioration after subtemporal decompression [3].…”

Section: Subtemporal Decompressionmentioning

confidence: 99%

“…Interestingly, Dandy noted that the craniectomy flaps of patients would often fluctuate between flat and tense within a period of minutes suggesting that the pressure elevations experienced by PTC patients were intermittent and often rapid in onset. Dandy felt that this rapid and intermittent rise in intracranial pressure was better explained by dynamic changes in the vasomotor vascular bed rather than more static problems such as CSF malabsorption or venous sinus occlusion [3].…”

Section: Introductionmentioning

confidence: 99%

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An update on the management of pseudotumor cerebri

Galgano¹,

Deshaies²

2013

Clinical Neurology and Neurosurgery

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Section: Subtemporal Decompressionmentioning

confidence: 77%

Section: Subtemporal Decompressionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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An update on the management of pseudotumor cerebri

Galgano¹,

Deshaies²

2013

Clinical Neurology and Neurosurgery

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“…These procedures are now infrequently used because of complications, which although rare include seizures, otorrhea, and subdural hematoma. However, long-term success has been reported70 and this procedure may be underutilized.…”

Section: Surgical Therapymentioning

confidence: 99%

Idiopathic Intracranial Hypertension

2010

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Idiopathic intracranial hypertension ((IIH) is characterized by increased cerebrospinal fluid pressure of unknown cause. It is predominantly a disease of women in the childbearing years. Although the cause of IIH remains obscure, it has become clear that loss of visual function is common and patients may progress to blindness if untreated. Diagnosis should adhere to the modified Dandy criteria and other causes of intracranial hypertension sought. IIH patient management should include serial perimetry and optic disc grading or photography. The proper therapy can then be selected and visual loss prevented or reversed. Although there are no evidence-based data to guide therapy, there is an ongoing randomized double-blind controlled treatment trial of IIH investigating diet and medical therapy.

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“…These procedures have largely been abandoned because of complications that include seizures, otorrhea, subdural hematoma, and postoperative visual deterioration. Long-term success has been reported, however [43].…”

Section: Surgical Therapymentioning

confidence: 99%

Idiopathic intracranial hypertension (Pseudotumor cerebri)

Wall

2008

Curr Neurol Neurosci Rep

105

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Idiopathic intracranial hypertension (IIH), also known as pseudotumor cerebri, is a disorder of elevated intracranial pressure of unknown cause. Patients present with daily headache, pulse-synchronous tinnitus, transient visual obscurations, papilledema with its associated visual loss, and diplopia from sixth nerve paresis. Many disease associations have been alleged, but few besides obesity, hypervitaminosis A and related compounds, steroid withdrawal, and female gender have been proven. Although absorption of cerebrospinal fluid (CSF) occurs through arachnoid granulations and extracranial lymphatics, outflow resistance is increased in IIH; therefore, intracranial pressure must increase for CSF to be absorbed. The mainstays of medical treatment are a reduced-sodium weight-reduction program and acetazolamide. If patients fail medical therapy, surgical procedures, most commonly optic nerve sheath fenestration and CSF shunting, are employed. The main morbidity of IIH is visual loss. This is present in most patients and can usually be reversed if recognized early in the course of the disease and treated.

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Surgical Treatment of Benign Intracranial Hypertension—Subtemporal Decompression Revisited

Cited by 33 publications

References 8 publications

An update on the management of pseudotumor cerebri

An update on the management of pseudotumor cerebri

Idiopathic Intracranial Hypertension

Idiopathic intracranial hypertension (Pseudotumor cerebri)

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