Surveillance of infection status of drug resistant Staphylococcus aureus in an Indian teaching hospital

Antimicrobial Resistance Pattern among Community-Acquired Gram-Positive and Gram-Negative Bacterial Bloodstream Isolates in India

“…This co-resistance could be mediated due to thickened cell wall, changes in cellular metabolism, and enhanced cell wall turnover that could interfere with the antimicrobial action of glycopeptides. Also, in case of S. aureus isolates, vancomycin, teicoplanin and linezolid reported high antimicrobial activity of 96.3%, 100% and 100% respectively; a finding similar to other studies [4][5][6][7][8][9][10]. On the contrary, reduced susceptibility was reported by Dubey et al (vancomycin,44.9%; teicoplanin, 44.7%; linezolid, 76.6%) [5].…”

Surveillance of multidrug resistant uropathogenic bacteria in hospitalized patients in Indian

“…Alike E. coli, S. aureus was a commensal of soft tissues and internal nares of nose, but methicillin resistant S. aureus (MRSA) strains have become resistant to the penicillin group of antibiotics due to ESBL genes. Not surprisingly, MRSA has slowly developed resistances to all other antibiotics of all major classes of the time, even many of which were never used for MRSA, raising its survival strength to withstand 23 antibiotics [36] . MRSA has now become MDR-MRSA and is considered as the superbug in the health domain.…”

“…MRSA has now become MDR-MRSA and is considered as the superbug in the health domain. This bacterium is the primary cause of suppurative infection and creates intimidatory clinical consternations mainly in surgical wound sites [36] .…”

A report on infection dynamics of inducible clindamycin resistance of Staphylococcus aureus isolated from a teaching hospital in India

“…Such events occur continually and independently with each pathogen. This is the mechanism of transformation of the harmless commensal S. aureus to the ghoulish, intractable, perilous and wily superbug MRSA [23] .…”