Symptomatic Apical Periodontitis of the Mandibular First Molar with the Accessory Canal in the Furcation Area Mimicking Furcation Perforation

Bolyachin, Alexei; Khabadze, Z. S.; Mordanov, Oleg; Gasbanov, M. A.; Tamaev, T. I.

doi:10.1155/2022/6324447

Cited by 3 publications

(4 citation statements)

References 24 publications

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“…No significant correlation was noted between lesion size and location in the present study; however, the majority of lesions were apical lesions in both jaws, which was expected, since the path of odontogenic infections is through the main canal to the apex [ 25 , 28 ]. In addition, a study of the prevalence of accessory canals in different parts of the teeth revealed that 74% of accessory canals are in the apical third, 15% are in the cervical third, and 11% are in the middle third; thus, higher frequency of apical lesions is justified [ 29 , 30 ]. Meng et al [ 10 ] evaluated the correlation of roots and endodontic lesions in the sagittal plane and reported that root and radicular cysts mostly had a centripetal relationship, and only a small number of cysts had horizontal expansion towards the palatal or buccal surface.…”

Section: Discussionmentioning

confidence: 99%

“…In the present study, the frequency of furcal and furcal to apical lesions was significantly higher in the mandible than maxilla, which was expected considering the root anatomy of mandibular and maxillary molars, since the roots are more divergent in the mandible and dentin thickness is thinner at the furcation site. Moreover, several accessory foramina are present in the furcation area of mandibular molars, which cannot be cleaned and shaped [ 30 ]; thus, the risk of developing furcal lesions is higher in the mandible.…”

Section: Discussionmentioning

confidence: 99%

“…Since mesial roots have greater curvature than distal roots [ 29 ], they are at higher risk of procedural errors such as strip perforation. Further, it has been demonstrated that 80% of mesial roots of mandibular first molars have an isthmus in the apical to the middle third of the root, which contains pulp tissue, and the cleaning and shaping of this area are highly difficult if not impossible; thus, it may remain and serve as a source of infection [ 29 , 30 ].…”

Section: Discussionmentioning

confidence: 99%

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Pattern of Endodontic Lesions of Maxillary and Mandibular Posterior Teeth: A Cone-Beam Computed Tomography Study

et al. 2022

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The pattern of expansion of endodontic lesions in the jaws has been less commonly addressed in the literature. For this reason, the aim of this study is to assess the pattern of endodontic lesions of maxillary and mandibular posterior teeth using cone-beam computed tomography (CBCT). This cross-sectional study was conducted on 317 endodontic lesions of posterior teeth on CBCT scans retrieved from a radiology center in Qazvin, Iran, from 2020 to 2022. Endodontic lesions were assessed on sagittal, coronal, and axial sections by an endodontist and dental student using the Romexis software. The largest lesion diameter was measured occluso-apically, mesiodistally, and buccolingually. Lesion size was analyzed based on age, gender, jaw, tooth type, and presence/absence of root filling by independent samples t-tests and a one-way Analysis Of Variannce (ANOVA). The largest diameter of lesions in the maxilla and mandible was recorded in the occluso-apical dimension followed by buccolingual and mesiodistal dimensions (p > 0.05). The pattern of lesions was the same in teeth with and without endodontic treatment, but it was significantly different in maxillary and mandibular endodontically treated teeth in the occluso-apical and buccolingual dimensions (p < 0.05). No significant correlation was noted with tooth type or jaw except for maxillary and mandibular first molar lesions, which were significantly different in the occluso-apical dimension (p < 0.05). Lesion size in all three dimensions was significantly greater in males than females (p < 0.05), and was the highest in the occluso-apical dimension in both genders. In the maxilla, the mean lesion size significantly decreased in the mesiodistal dimension with age (p < 0.05). In conclusion, the largest lesion diameter in the maxilla and mandible was found in the occluso-apical dimension, indicating the role of bone density in the pattern of lesions.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Pattern of Endodontic Lesions of Maxillary and Mandibular Posterior Teeth: A Cone-Beam Computed Tomography Study

et al. 2022

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“…In the case of the structure of teeth, their sensitive part, which is the pulp and the root canal system, is an intact place to which both pathogens and commensal bacteria in the oral cavity should not have access [ 6 , 7 ]. When the dentinal barriers are damaged by caries, fissures, or trauma, both opportunistic and pathogenic microorganisms gain access to the pulp chamber and root canal system to which they should never have access [ 8 , 9 , 10 ]. This situation could lead to harmful effects such as inflammation, pulp necrosis, and periodontitis [ 11 , 12 , 13 ].…”

Section: Introductionmentioning

confidence: 99%

Root Canal Infection and Its Impact on the Oral Cavity Microenvironment in the Context of Immune System Disorders in Selected Diseases: A Narrative Review

Sobieszczański

Mertowski

Sarna‐Boś

et al. 2023

JCM

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The oral cavity has a specific microenvironment, and structures such as teeth are constantly exposed to chemical and biological factors. Although the structure of the teeth is permanent, due to exposure of the pulp and root canal system, trauma can have severe consequences and cause the development of local inflammation caused by external and opportunistic pathogens. Long-term inflammation can affect not only the local pulp and periodontal tissues but also the functioning of the immune system, which can trigger a systemic reaction. This literature review presents the current knowledge on root canal infections and their impact on the oral microenvironment in the context of immune system disorders in selected diseases. The result of the analysis of the literature is the statement that periodontal-disease-caused inflammation in the oral cavity may affect the development and progression of autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus, or Sjogren’s syndrome, as well as affecting the faster progression of conditions in which inflammation occurs such as, among others, chronic kidney disease or inflammatory bowel disease.

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The Features of Chronic Periodontitis, Complicated by Furcation Defects. Causes of Defects. Literature Review

Khabadze

Gasbanov²,

Bolyachin³

et al. 2022

Actual Problems in Dentistry

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Periodontal tissue diseases remain one of the urgent problems in modern dentistry due to their high prevalence and the number of complications after treatment. The prolonged asymptomatic course of chronic forms of periodontitis causes an increase in destructive processes not only in periodontal tissues and alveolar bone, but also contributes to the resorption of cementum and root dentin, which causes the formation of bone pockets, called furcation defects. Involvement in the pathological process of the zone of divergence of the roots significantly complicates the course of the disease, diagnosis, treatment and prognosis. The complexity of diagnosis, the variability of treatment methods depending on the overall clinical picture and the high percentage of tooth loss when the furcation area is involved in the pathological process determine the relevance of the research topic. Purpose: to study the features of chronic periodontitis complicated by furcation defects and to identify the main causes of these defects. Materials and Methods: the analysis of domestic and foreign publications placed in the eLibrary and PubMed databases was carried out by keywords. Results. The analysis of literature data on the features of chronic periodontitis complicated by furcation defects is carried out, the issues of the etiology of this pathology are considered. Conclusion. It is shown that the treatment of chronic apical periodontitis complicated by furcation defects is one of the important problems of dentistry, which is determined by the significant prevalence of the disease and the incidence of complications after treatment. The most common etiological factor in furcation lesions is microbial invasion, which causes an inflammatory process, the progression of which leads to bone resorption in the area of the furcation arch of the teeth. Also, the etiological factors of periodontal pathogenic genesis, endodontic lesions, fractures, iatrogenic factors, as well as anatomical features contribute to the involvement of furcation.

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Symptomatic Apical Periodontitis of the Mandibular First Molar with the Accessory Canal in the Furcation Area Mimicking Furcation Perforation

Cited by 3 publications

References 24 publications

Pattern of Endodontic Lesions of Maxillary and Mandibular Posterior Teeth: A Cone-Beam Computed Tomography Study

Pattern of Endodontic Lesions of Maxillary and Mandibular Posterior Teeth: A Cone-Beam Computed Tomography Study

Root Canal Infection and Its Impact on the Oral Cavity Microenvironment in the Context of Immune System Disorders in Selected Diseases: A Narrative Review

The Features of Chronic Periodontitis, Complicated by Furcation Defects. Causes of Defects. Literature Review

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