Synthesis and characterization of the Anderson–Evans tungstoantimonate [Na₅(H₂O)₁₈{(HOCH₂)₂CHNH₃}₂][SbW₆O₂₄]

Abstract

A novel tungstoantimonate, [Na5(H2O)18{(HOCH2)2CHNH3}2][SbVWVI 6O24] (SbW6 ), was synthesized from an aqueous solution and structurally characterized by single-crystal X-ray diffraction, which revealed C2/c symmetry. The structure contains two serinol [(HOCH2)2CHNH3]+ and five Na+ cations, which are octahedrally surrounded by 18 water molecules, and one [SbVWVI 6O24]7− anion. The serinol molecules also play a critical role in the synthesis by acting as a mild buffering agent. Each of the WVI and SbV ions is six-coordinated and displays a distorted octahedral motif. A three-dimensional supramolecular framework is formed via hydrogen-bonding interactions between the tungstoantimonates and cations. Powder X-ray diffraction, elemental analysis, thermogravimetric analysis and IR spectroscopy were performed on SbW6 to prove the purity, to identify the water content and to characterize the vibrational modes of the crystallized phase.

Heteroelements in polyoxometalates: a study on the influence of different group 15 elements on polyoxometalate formation

“…Finally, we performed the same experiments using potassium hexahydroxoantimonate(V) as an antimony precursor, which has been reported previously as a precursor for incorporating Sb(V) into a POM structure. 45 At the pH value of 5, we observed the formation of the Anderson-Evans-type anion [SbW 6 O 24 ] 7− , which was first identified by singly crystal XRD (Fig. 14).…”

“…Finally, we performed the same experiments using potassium hexahydroxoantimonate( v ) as an antimony precursor, which has been reported previously as a precursor for incorporating Sb( v ) into a POM structure. 45…”

Demethylnobiletin ameliorates cerebral ischemia‐reperfusion injury in rats through Nrf2/HO‐1 signaling pathway

“…Decades of preclinical researches have proved the potential benefits of neuroprotectants in stroke models. They are involved in modulating intracellular signaling pathways to ameliorate poststroke edema, excitatory toxicity, OS, inflammation, and apoptosis 26 . In our study, TTC staining and mNSS were used to confirm the effect of DN on decreasing cerebral infarction area and promoting neurological function recovery in MCAO rats.…”

The Effect of Tuberculosis Infection on Pancreatic Beta-Cell Function in Patients with Type 2 Diabetes Mellitus

“…Tuberculosis is a common chronic disease threatening human health caused by Mycobacterium tuberculosis infection, which mainly affects the glucose metabolism of diabetic patients and aggravates the condition; at the same time, fever and other toxic symptoms caused by tuberculosis infection can lead to impaired pancreatic islet β-cell function, which reduces the function of insulin receptor and affects the secretion function of pancreatic islets [9]. Related studies have shown that gender and duration of diabetes are risk factors for PTB combined with T2DM [10] [11], and that men and long duration of disease are at higher risk of developing TB and diabetes. Therefore, this study excluded the effect of gender and disease duration, (in order to avoid the influence of the difference between the two groups of gender and the course of diabetes on the relationship between the disease and the factors, so as to more truly reflect the relationship between the research factors and the disease, therefore, this study adopted a 1:1 matching method according to gender and the course of diabetes, which can not only control the influence of confounding factors, but also improve the efficiency of the study and ensure the comparability between the study group and the control group.)…”