Temperature regulation during severe exercise with some observations on effects of skin wetting

Davies, C. T. M.; Brotherhood, J. R.; Zeidifard, E.

doi:10.1152/jappl.1976.41.5.772

Cited by 56 publications

(34 citation statements)

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“…Numerous investigators (Davies 1979;Davies et al 1976;Saltin et al 1966) have reported that the magnitude of core temperature elevation during muscular exercise is coupled to the relative exercise intensity. This relationship has been…”

Section: Discussionmentioning

confidence: 99%

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Influence of heat stress and acclimation on maximal aerobic power

Sawka

Young

Cadarette

et al. 1985

Europ. J. Appl. Physiol.

115

108

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Section: Discussionmentioning

confidence: 99%

“…reported for relative exercise intensities up to 85% V0 2 max (Davies 1979) as well as in moderate and hot environmental conditions (Davies et al 1976). If heat acclimation did enable individuals to achieve higher '02 max during heat stress, then individuals would be at lowered relative intensities at a given submaximal oxygen uptake level.…”

Section: N1mentioning

confidence: 99%

Influence of heat stress and acclimation on maximal aerobic power

Sawka

Young

Cadarette

et al. 1985

Europ. J. Appl. Physiol.

115

108

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“…Median VO 2max was 44 mL·kg −1 ·min −1 (39)(40)(41)(42)(43)(44)(45)(46)(47)(48)(49). Maximal HR achieved during the determination of VO 2max was significantly lower than maximal HR recorded during the run (189 ± 9 versus 199 ± 9, respectively, p < 0.001).…”

Section: Vo 2maxmentioning

confidence: 99%

“…Mean HR recorded during VT1 was 157 ± 17 bpm, corresponding to 78 ± 8% of maximal HR recorded during the run. Subjects with VO 2max > 43 mL·kg −1 ·min −1 exhibited the lower BFM, estimated either by skinfold thickness (34 mm [27][28][29][30][31][32][33][34][35][36][37][38][39][40] versus 50 mm and lowest BFM, estimated either by skinfold thickness or impedancemetry. However, the velocity was only associated with VO 2max in a multivariate analysis including both variables.…”

Section: Vo 2maxmentioning

confidence: 99%

Thermoregulatory Response to Exercise After Exertional Heat Stroke

Sagui¹,

beighau²,

Jouvion

et al. 2017

Military Medicine

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Background: After one episode of exertional heat stroke (EHS), risk factors must be identified to determine the potential for subsequent episodes. One of these risk factors, core body temperature (T co ) kinetics during strenuous exercise, may be a surrogate marker suggestive of impaired thermoregulation. This study aimed to determine the kinetics of increases in T co among military subjects who had a history of EHS. Methods: Forty subjects (38 males, mean age 28.4 ± 4.9 years, mean body mass index 24.9 ± 2.4) who had a history of EHS ran 8 km in full combat gear with continuous monitoring of T co and heart rate. The run was a qualifying event for military service. T co was assessed using an ingestible sensor (Cortemp HQ Inc., Palmetto, Florida). Maximum oxygen uptake (VO 2max ) was measured on the day before the run. Findings: The mean performance time for the run was 44.6 ± 6.6 minutes achieved under mild climatic conditions. No neurological impairment was observed. The mean maximum T co was 39.9 ± 0.5°C. On the basis of T co during the last 10 minutes of running, two T co profiles were identified: increased T co (T co increase > 0.5°C) and plateaued T co . Neither profile depended on initial, mid-run, or maximal T co , VO 2max , speed running, body surface area or body fat mass. Discussion: Subjects who had a history of EHS exhibited different T co profiles at the end of an 8-km run. Laboratory studies will be necessary to identify the mechanisms underlying these profiles; future longitudinal studies can determine whether a T co increase >0.5°C during the last 10 minutes is a risk factor for EHS recurrence.

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“…However, it is the metabolic rate that determines sweat rate and body temperature during exercise (8,17,72,80,90,103). Thus heavier runners will sweat heavily only if they also run fast and maintain high metabolic rates during exercise; in reality, heavier runners tend to run slower than lighter runners, thus their sweat rates may not be nearly as great as those predicted by Wyndham and Strydom (102).…”

Section: Fluid Balance During Prolonged Exercisementioning

confidence: 99%

The Hyponatremia of Exercise

Noakes¹

1992

International Journal of Sport Nutrition

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The hyponatremia of exercise may exist in symptomatic and asymptomatic forms. Symptomatic hyponatremia is usually characterized by severe alterations in cerebral function including coma and grand ma1 seizures; it develops especially in less competitive athletes who have maintained high rates of fluid intake during endurance events lasting at least 5 hours. The hyponatremia becomes symptomatic when the volume of excess fluid retained exceeds 2 to 3 liters. The etiology of the condition is unknown. Possibly as many as three or more pathologies (abnormal fluid retention possibly due to inappropriate ADH secretion, abnormal regulation of the extracellular fluid volume, translocation of sodium into a "third space") must be present for symptomatic hyponatremia to develop. The avoidance of overhydration would appear to be the only certain way that susceptible individuals can prevent symptomatic hyponatremia. Sodium chloride containing solutions ingested in physiologically significant concentrations would likely prevent a possible "third space" effect.A belief widely propagated in the scientific literature is that fluid losses during prolonged exercise are large and that progressive dehydration is an inevitable consequence (2,3,5,(34)(35)(36)(38)(39)(40)48,54,78,79,87,95,101,102). Indeed some authors have estimated that fluid losses can be as much as 4 1 per hour during exercise in the heat (5,43,79). Since maximum sustainable rates of fluid delivery to the intestine even during exercise of short duration are of the order of only 1 to 2 1 per hour (60), it follows that even if maximum rates of intestinal water absorption can match such high rates of fluid ingestion, progressive dehydration would be an inevitable consequence of prolonged exercise were such high sweat rates maintained.Progressive dehydration is believed to cause hyperthermia in proportion to the fluid deficit (39,43,101,102) and, when extreme, is considered to be the principal cause of the exercise-associated collapse that has become a common feature especially of ultradistance running and triathlon events (2, 36, 39,43,48, 66-68, 70, 78, 83, 85, 87, 91, 95). Therefore persons participating i n prolonged exercise have been encouraged to drink as much as possible to avoid these potentially serious complications, particularly heatstroke and other forms of heat Timothy D. Noakes is with the

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Temperature regulation during severe exercise with some observations on effects of skin wetting

Cited by 56 publications

References 0 publications

Influence of heat stress and acclimation on maximal aerobic power

Influence of heat stress and acclimation on maximal aerobic power

Thermoregulatory Response to Exercise After Exertional Heat Stroke

The Hyponatremia of Exercise

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