Tentorial dural arteriovenous fistula presenting symptoms due to mass effect on the dilated draining vein: case report

Dural arteriovenous fistulas draining into the petrosal vein or bridging vein of the medulla: possible homologs of spinal dural arteriovenous fistulas

“…We excluded DAVFs located in remote regions that drained into posterior fossa veins secondary to thrombosis of their "natural" outlets, in particular cavernous sinus or transversesigmoid lesions with venous occlusions. In the previously published literature, we found 30 cases of petrosal vein DAVFs 4,5,[7][8][9]13,16,21,30,33,[36][37][38]41,45,48,52,55,56,59,60 and 35 cases of medulla bridging vein DAVFs, 1,11,14,[17][18][19][20][22][23][24][25]31,32,35,39,40,43,46,50,51,53,58,61 all of which were well described with clinical and angiographic information. These previously reported cases were analyzed together, with special attention to their clinical characteristics.…”

Complications Related to Percutaneous Transarterial Embolization of Intracranial Dural Arteriovenous Fistulas in 40 Patients

“…2,[19][20][21][22][23][24] High-risk fistulas typically present with hemorrhage or progressive neurologic deficits. 13,[25][26][27][28][29][30][31][32][33][34][35][36] Most DAVFs can be successfully managed with endovascular techniques. Transarterial Onyx (ev3, Irvine, Calif) embolization is now an established method of treatment of DAVFs.…”

Endovascular treatment resolves non-hemorrhagic brainstem dysfunction due to tentorial dural AV fistula

“…13 The mechanism of brainstem dysfunction may be related to arterial steal phenomena, brainstem venous congestion or compression due to dilated veins or cerebellar edema. 8,12,13 In our patient, gait instability was attributed to cerebellar edema especially of the left lobe and vermis ( Fig. 1), while double vision was probably related to brainstem compression resulting from the edema.…”

“…Specific CNS manifestations may be the result of venous ischemia, venous mass effect, venous rupture or venous thrombosis due to passive venous hypertension, congestion or both. 8 Many non-hemorrhagic neurological deficits due to tDAVFs have been reported, including gait instability, bruit, personality changes, depression, trigeminal neuralgia, syncopal events, slurred speech, cranial nerve VII weakness, 3 visual symptoms (including bilateral proptosis, bilateral episcleral and retinal venous congestion, optic disc palor, quadrantanopia and concentric narrowing of the visual field), 9 hemifacial spasm, 10 progressive myelopathy, 11 hemisensory disturbance, 12 and brainstem dysfunction. 13 The mechanism of brainstem dysfunction may be related to arterial steal phenomena, brainstem venous congestion or compression due to dilated veins or cerebellar edema.…”