2003
DOI: 10.1038/sj.leu.2403107
|View full text |Cite|
|
Sign up to set email alerts
|

The anticancer plant stress-protein methyl jasmonate induces activation of stress-regulated c-Jun N-terminal kinase and p38 protein kinase in human lymphoid cells

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
3
1
1

Citation Types

2
24
0

Year Published

2004
2004
2019
2019

Publication Types

Select...
9
1

Relationship

0
10

Authors

Journals

citations
Cited by 29 publications
(26 citation statements)
references
References 8 publications
2
24
0
Order By: Relevance
“…Methyl jasmonate induced death in breast and prostate carcinoma cells, as well as in melanoma, lymphoma, and leukemia cells (4). Furthermore, we determined that jasmonates are capable of killing cancer cells in a manner independent of cellular mRNA transcription, protein translation (5), and p53 expression. 3 Finally, methyl jasmonate significantly increased the life span of lymphomabearing mice (4).…”
Section: Introductionmentioning
confidence: 92%
“…Methyl jasmonate induced death in breast and prostate carcinoma cells, as well as in melanoma, lymphoma, and leukemia cells (4). Furthermore, we determined that jasmonates are capable of killing cancer cells in a manner independent of cellular mRNA transcription, protein translation (5), and p53 expression. 3 Finally, methyl jasmonate significantly increased the life span of lymphomabearing mice (4).…”
Section: Introductionmentioning
confidence: 92%
“…A recent report shows that MJ induces activation of stress-regulated c-Jun N-terminal kinase and p38 protein kinase in lymphoid cells. 31 Further experimentation is necessary to understand the role of MAPKs on anticancer action of MJ.…”
Section: Discussionmentioning
confidence: 99%
“…JAs also are implicated in the control of plant responses to abiotic stimuli such as mechanical stress (Weiler et al, 1993), salt stress (Dombrowski, 2003), UV irradiation (Conconi et al, 1996), and ozone exposure (Rao et al, 2000). The ability of JAs to regulate gene expression in plant, insect (Li et al, 2002c), and mammalian (Rotem et al, 2003) cells indicates that some components of this lipid-based signaling system may be conserved in diverse biological systems.…”
Section: Introductionmentioning
confidence: 99%