The association between exposure to air pollutants including PM10, PM2.5, ozone, carbon monoxide, sulfur dioxide, and nitrogen dioxide concentration and the relative risk of developing STEMI: A case-crossover design

Akbarzadeh, Mohammad Ali; Khaheshi, Isa; Sharifi, Amirsina; Yousefi, Nazila; Naderian, Mohammadreza; Namazi, Mohammad Hasan; Safi, Morteza; Vakili, Hossein; Saadat, Habibollah; Parsa, Saeed; Nickdoost, Negin

doi:10.1016/j.envres.2017.11.020

Cited by 65 publications

(26 citation statements)

References 35 publications

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“…PM 10 also had detrimental effects on cardiovascular diseases. A 10 µg/m 3 increase in PM 10 concentration was related to a 10.10% increase in the incidence of ST-elevation myocardial infarction 29 . This increase in the incidence of cardiovascular disease could be related to the risk of Bell's palsy.…”

Section: Discussionmentioning

confidence: 89%

Air pollution by NO2 is associated with the risk of Bell’s palsy: A nested case-controlled study

Kim

Min

Choi

et al. 2020

Sci Rep

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this study investigated the relationship of weather and air pollution with the onset of Bell's palsy. the Korean Health insurance Review and Assessment Service-national Sample cohort (HiRA-nSc) data from 2002 through 2013 were used. The 3,935 Bell's palsy patients were matched with 15,740 control participants. the meteorological data, including daily mean temperature (°c), daily mean highest temperature (°C), daily mean lowest temperature (°C), daily mean temperature difference (°C), relative humidity (%), spot atmospheric pressure (hpa), sulfur dioxide (So 2) (ppm), nitrogen dioxide (no 2) (ppm), ozone (o 3) (ppm), carbon monoxide (co) (ppm), and pM 10 (particulate matter ≤ 10 μg/m 3) for 60 days, 30 days, 14 days, 7 days, and 3 days prior to the index date were analyzed for Bell's palsy cases and controls. conditional logistic regression analysis was used to estimate the odds ratios (oRs) of the association between the meteorological data and Bell's palsy. the mean no 2 and pM 10 concentrations for 60 days were higher, while that of O 3 was lower in the Bell's palsy group than in the control group (both p < 0.001). The Bell's palsy group showed 16.63-fold higher odds of NO 2 for 60 days (0.1 ppm) than the control group (95% CI = 10.18-27.16, P < 0.001). The ORs of PM 10 , and o 3 for 60 days showed inconsistent results according to the included variables. Bell's palsy was related to high concentrations of no 2 .

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Section: Discussionmentioning

confidence: 89%

Air pollution by NO2 is associated with the risk of Bell’s palsy: A nested case-controlled study

Kim

Min

Choi

et al. 2020

Sci Rep

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“…2 Overall analyses of the effect on the risk of MI hospitalizations associated with a 10 μg/m3 increase in PM 2.5 hospitalization for myocardial infarction was 2,250,473. The largest number of participants was 452,343, which belonged to the study by Milojevic et al, (2014), and the smallest number of participants was 208, which was reported by Akbarzadeh et al, (2018). Considering Berglind et al (2010), the research was conducted in three cities (Boston, Seattle, and Augsburg), the lag averaging time was 2 h, and the adjusted odds ratio for the PM 2.5 pollutant was applied in the analysis [39].…”

Section: Resultsmentioning

confidence: 99%

Association between PM2.5 and risk of hospitalization for myocardial infarction: a systematic review and a meta-analysis

et al. 2020

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Background: It is generally assumed that there have been mixed results in the literature regarding the association between ambient particulate matter (PM) and myocardial infarction (MI). The aim of this meta-analysis was to explore the rate of short-term exposure PM with aerodynamic diameters ≤2.5 μm (PM 2.5) and examine its potential effect(s) on the risk of MI. Methods: A systematic search was conducted on databases like PubMed, Scopus, Web of Science, and Embase with components: "air pollution" and "myocardial infarction". The summary relative risk (RR) and 95% confidence intervals (95%CI) were also calculated to assess the association between the PM 2.5 and MI. Results: Twenty-six published studies were ultimately identified as eligible candidates for the meta-analysis of MI until Jun 1, 2018. The results illustrated that a 10-μg/m 3 increase in PM 2.5 was associated with the risk of MI (RR = 1.02; 95% CI 1.01-1.03; P ≤ 0.0001). The heterogeneity of the studies was assessed through a random-effects model with p < 0.0001 and the I 2 was 69.52%, indicating a moderate degree of heterogeneity. We also conducted subgroup analyses including study quality, study design, and study period. Accordingly, it was found that subgroups time series study design and high study period could substantially decrease heterogeneity (I 2 = 41.61, 41.78). Conclusions: This meta-analysis indicated that exposureresponse between PM 2.5 and MI. It is vital decision makers implement effective strategies to help improve air pollution, especially in developing countries or prevent exposure to PM 2.5 to protect human health.

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“…Short and long-term exposures to air pollutants (especially PM 2.5 , but also PM 10 and NO 2 ) have been found to be related to an increased risk of segment elevation myocardial infarction (STEMI) [ 42 , 43 ]. Older people are generally considered to be more susceptible to the effects of air pollution because of the gradual decline in physiological processes over time as well as the presence of underlying cardiovascular risk factors (e.g., obesity, metabolic syndrome) or pre-existing coronary artery disease, chronic lung disease, or heart failure [ 44 , 45 ].…”

Section: Possible Causal Links Between Covid-19 Infection and Amimentioning

confidence: 99%

“Acute Myocardial Infarction in the Time of COVID-19”: A Review of Biological, Environmental, and Psychosocial Contributors

Gorini

Chatzianagnostou

Mazzone

et al. 2020

IJERPH

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Coronavirus disease 2019 (COVID-19) has quickly become a worldwide health crisis.Although respiratory disease remains the main cause of morbidity and mortality in COVID patients,myocardial damage is a common finding. Many possible biological pathways may explain therelationship between COVID-19 and acute myocardial infarction (AMI). Increased immune andinflammatory responses, and procoagulant profile have characterized COVID patients. All theseresponses may induce endothelial dysfunction, myocardial injury, plaque instability, and AMI.Disease severity and mortality are increased by cardiovascular comorbidities. Moreover, COVID-19has been associated with air pollution, which may also represent an AMI risk factor. Nonetheless,a significant reduction in patient admissions following containment initiatives has been observed,including for AMI. The reasons for this phenomenon are largely unknown, although a real decreasein the incidence of cardiac events seems highly improbable. Instead, patients likely may presentdelayed time from symptoms onset and subsequent referral to emergency departments because offear of possible in-hospital infection, and as such, may present more complications. Here, we aim todiscuss available evidence about all these factors in the complex relationship between COVID-19and AMI, with particular focus on psychological distress and the need to increase awareness ofischemic symptoms.

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The association between exposure to air pollutants including PM10, PM2.5, ozone, carbon monoxide, sulfur dioxide, and nitrogen dioxide concentration and the relative risk of developing STEMI: A case-crossover design

Cited by 65 publications

References 35 publications

Air pollution by NO2 is associated with the risk of Bell’s palsy: A nested case-controlled study

Air pollution by NO2 is associated with the risk of Bell’s palsy: A nested case-controlled study

Association between PM2.5 and risk of hospitalization for myocardial infarction: a systematic review and a meta-analysis

“Acute Myocardial Infarction in the Time of COVID-19”: A Review of Biological, Environmental, and Psychosocial Contributors

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