SummaryTraumatic tricuspid regurgitation is a rare and progressive disease. Early diagnosis and surgical valve repair are very important. A 57-year-old male was referred to our hospital with a history of blunt chest trauma. Three-dimensional echocardiography showed severe tricuspid regurgitation and demonstrated two main anterior leaflet chordaes of the tricuspid valve rupture and the whole anterior leaflet prolapsed. The diagnosis was severe tricuspid regurgitation due to leaflet chordae rupture secondary to blunt chest trauma. Surgical repair of the tricuspid valve was performed in this patient. At 3-month follow-up, the right ventricle was decreased in size with significantly improved right ventricular function. The signs and symptoms of right heart failure were relieved. In this case, 3-dimensional transthoracic echocardiography enabled fast and non-invasive evaluation of the spatial destruction of the tricuspid valve and subvalvular apparatus to assist in the planning of valve repair. (Int Heart J 2017; 58: 451-453) Key words: Tricuspid valve prolapse, Valve repair, Right heart failure, Transthoracic T raumatic tricuspid regurgitation (TR) is a rare cardiovascular complication of blunt chest trauma. 1) When feasible, tricuspid valve repair is preferable to valve replacement. We report a clinical case in which we describe the usefulness of 3-dimensional (3D) transthoracic echocardiography (TTE) for the diagnosis and detailed preoperative assessment of traumatic TR to assist in planning the surgical procedure.
Case ReportA 57-year-old male was referred to our hospital with increasing occurrence of palpitations, dyspnea on exertion, and fatigue. Two months prior to presentation, he suffered from a punch in the chest by door panel, including a minor rib fracture. Physical examination revealed the following: blood pressure 118/72 mmHg, heart rate 103 beats/minute, temperature 36.4°C, and respiratory rate 20 breaths/minute. Physical examination revealed hepatomegaly, peripheral edema, and a grade 3-4/6 systolic murmur at the right lower sternal edge. The initial laboratory examination demonstrated that C-reactive protein (CRP) was 26.6 mg/L (n < 8.0 mg/L), troponin I 7.2 ng/ mL (n < 0.033 ng/mL), and creatine kinase 291 U/L (n = 38-174 U/L).2D TTE revealed that the right atrium and right ventricle were dilated. RV fractional area change (FAC) was 20% (Figure 1A). Tricuspid annular plane systolic excursion (TAPSE) was 11 mm. The right ventricular systolic function was impaired. Color Doppler echocardiography demonstrated the presence of severe tricuspid regurgitation (Figure 2A). The precise mechanism for severe TR was not clearly defined. It might be functional TR with dilatation of the right heart or primary tricuspid valve pathology caused by trauma, infective endocarditis, or congenital heart disease. To better define the anatomy of the tricuspid valve, 3D TTE was performed. 3D datasets were acquired from both the right ventricle focused apical 4-chamber view and the right ventricular inflow tract view. A r...